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Muslum Kuzu

Researcher at Karabük University

Publications -  33
Citations -  482

Muslum Kuzu is an academic researcher from Karabük University. The author has contributed to research in topics: Chemistry & Carbonic anhydrase. The author has an hindex of 9, co-authored 27 publications receiving 293 citations. Previous affiliations of Muslum Kuzu include Ağrı İbrahim Çeçen University & Atatürk University.

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Morin attenuates doxorubicin-induced heart and brain damage by reducing oxidative stress, inflammation and apoptosis.

TL;DR: The regulatory role of morin in signal transduction in the brain tissue was assigned with the determination of amount of acetylcholinesterase (AChE), and its healing effect on the central nervous system was determined with imuinohistochemical detection of glial fibrillar acidic protein (GFAP) level.
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Protective Effect of Hesperidin on Sodium Arsenite-Induced Nephrotoxicity and Hepatotoxicity in Rats

TL;DR: In this study, it was seen that HSP showed a protective effect against SA-induced kidney and liver toxicity and reduced apoptosis, oxidative stress, inflammation, and oxidative DNA damage significantly in SA- induced kidney and Liver tissues depending on dose.
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Protective effect of morin on doxorubicin-induced hepatorenal toxicity in rats.

TL;DR: Morin prevented tissue damage in liver and kidney tissues caused by DOX and prevented the increase in DOX-induced Bax expression and decrease in Bcl-2 level, AQP-2 and nephrin expression, according to histopathological examination.
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An analysis of expression patterns of genes encoding proteins with catalytic activities.

TL;DR: The 662 expression patterns discussed here comprised gene products with activities associated with catalysis, and it was revealed that a significant number of genes encoding housekeeping functions such as biosynthesis and catabolism were expressed regionally, so they could be used as tissue-specific gene markers.
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Attenuation of sodium arsenite-induced cardiotoxicity and neurotoxicity with the antioxidant, anti-inflammatory, and antiapoptotic effects of hesperidin

TL;DR: HES co-treatment had a protective effect on SA-induced heart and brain toxicity in rats, and it was determined that SA damaged tissue architecture, and as a result of immunohistochemical examination, it increased cardiac Bcl-2-associated X protein (Bax) and cerebral fibrillary acidic protein (GFAP) expression.