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Naila Rasheed

Researcher at Qassim University

Publications -  43
Citations -  1053

Naila Rasheed is an academic researcher from Qassim University. The author has contributed to research in topics: Dopaminergic & Oxidative stress. The author has an hindex of 20, co-authored 40 publications receiving 869 citations. Previous affiliations of Naila Rasheed include Central Drug Research Institute.

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Alterations in monoamine levels and oxidative systems in frontal cortex, striatum, and hippocampus of the rat brain during chronic unpredictable stress

TL;DR: The results suggest that the perturbed monoamine levels could interact with the oxidative load during chronic unpredictable stress, and have implications for pharmacological interventions targeting both central monoamines and cellular antioxidants as a potential stress management strategy for protecting against central stress-induced disorders.
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Enhanced recognition of reactive oxygen species damaged human serum albumin by circulating systemic lupus erythematosus autoantibodies

TL;DR: The binding characteristics of SLE autoantibodies with native and √OH damaged HSA were assessed and the increase in total serum protein carbonyl levels in the SLE patients was largely due to an increase in oxidized albumin.
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MicroRNA-26a-5p regulates the expression of inducible nitric oxide synthase via activation of NF-κB pathway in human osteoarthritis chondrocytes.

TL;DR: This is the first report that shows hsa-miR-26a-5p is a direct regulator of iNOS expression in human chondrocytes and may be an important regulator of human cartilage homeostasis and a new target for OA therapy.
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Differential response of central dopaminergic system in acute and chronic unpredictable stress models in rats.

TL;DR: In insights into the differential regional response of dopaminergic system during AS and CUS are provided, neurochemical and behavioral effects of D1 agonist pretreatment suggest specific modulatory role of D2-like receptors under such stressful episodes.
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Epigallocatechin-3- O -gallate modulates global microRNA expression in interleukin-1β-stimulated human osteoarthritis chondrocytes: potential role of EGCG on negative co-regulation of microRNA-140-3p and ADAMTS5

TL;DR: The data indicate that the potential of EGCG in OA chondrocytes may be related to its ability to globally inhibit inflammatory response via modulation of miRNAs expressions, and provides an important insight into the molecular basis of the reported anti-arthritic effects.