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Nancy L. Miller
Researcher at University of Texas Medical Branch
Publications - 16
Citations - 1342
Nancy L. Miller is an academic researcher from University of Texas Medical Branch. The author has contributed to research in topics: Bone disease & Parenteral nutrition. The author has an hindex of 14, co-authored 16 publications receiving 1322 citations. Previous affiliations of Nancy L. Miller include University of Colorado Denver & Tulane University.
Papers
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Journal ArticleDOI
Bone Aluminum and Histomorphometric Features of Renal Osteodystrophy
Anthony B. Hodsman,Donald J. Sherrard,Allen C. Alfrey,Susan M. Ott,Arnold S. Brickman,Nancy L. Miller,Norma A. Maloney,Jack W. Coburn +7 more
TL;DR: Findings are consistent with but do not prove the hypothesis that aluminum plays a pathogenic role in dialysis osteomalacia; the mechanism by which aluminum accumulates remains unknown.
Journal ArticleDOI
Evidence of aluminum loading in infants receiving intravenous therapy.
Aileen B. Sedman,Aileen B. Sedman,Aileen B. Sedman,Gordon L. Klein,Gordon L. Klein,Gordon L. Klein,Russell J. Merritt,Russell J. Merritt,Russell J. Merritt,Nancy L. Miller,Nancy L. Miller,Nancy L. Miller,Kim O. Weber,Kim O. Weber,Kim O. Weber,William L. Gill,William L. Gill,William L. Gill,Harish Anand,Harish Anand,Harish Anand,Allen C. Alfrey,Allen C. Alfrey,Allen C. Alfrey +23 more
TL;DR: It is concluded that infants receiving intravenous therapy have aluminum loading, which is reflected in increased urinary excretion and elevated concentrations in plasma and bone.
Journal ArticleDOI
Aluminum loading during total parenteral nutrition.
Gordon L. Klein,A C Alfrey,Nancy L. Miller,Donald J. Sherrard,T K Hazlet,Marvin E. Ament,Jack W. Coburn +6 more
TL;DR: Parenteral loading with A1 increases tissue A1, particularly in bone, and whether A1 accumulation contributes to bone disease remains unclear, but the prolonged use of casein in total parenterals nutrition solutions may be inadvisable.
Journal ArticleDOI
Evaluation of body magnesium stores.
TL;DR: It would appear from these results that bone and extracellular fluid magnesium are the major magnesium pools in man increased during magnesium excess and decreased during magnesium depletion.
Journal ArticleDOI
Bone Magnesium Pools in Uremia
Allen C. Alfrey,Nancy L. Miller +1 more
TL;DR: In vitro studies of bone magnesium pools suggest that surface magnesium should rapidly reflect changes in serum magnesium levels, whereas, the deeper magnesium pool is probably deposited at time of bone formation with mobilization being dependent upon the resorptive processes.