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Nanlin Yin

Researcher at Chongqing Medical University

Publications -  5
Citations -  26

Nanlin Yin is an academic researcher from Chongqing Medical University. The author has contributed to research in topics: Medicine & Microbiome. The author has an hindex of 1, co-authored 5 publications receiving 4 citations.

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Neonatal hair profiling reveals a metabolic phenotype of monochorionic twins with selective intrauterine growth restriction and abnormal umbilical artery flow.

TL;DR: It is demonstrated that the hair metabolite profiles of the different sIUGR subclinical forms were associated with the averaged fetal growth rate after 28 weeks of gestation but not with birthweight.
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Identification and Characterization of Circular RNA as a Novel Regulator and Biomarker in Preterm Birth.

TL;DR: Bioinformatics analysis revealed that the top 20 circRNAs competitively bind 68 miRNAs, thereby regulating 622 mRNAs mainly related to immunity, inflammation, and nerve activity, which may ultimately contribute to the occurrence of PTB.
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Identification of the correlations between interleukin-27 (IL-27) and immune-inflammatory imbalance in preterm birth.

TL;DR: In this paper, the role of interleukin-27 (IL-27), an immunomodulatory factor, in preterm birth and its associated mechanisms was identified, which may provide a novel strategy for the prevention and treatment of PTB.
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IL-27 Mediates Pro-Inflammatory Effects via the ERK Signaling Pathway During Preterm Labor.

TL;DR: In this paper, the authors established a Lipopolysaccharide (LPS)-stimulated, infection-induced, preterm mouse model based on wild-type C57BL/6 mice and WSX-1-/-C57BL /6 mice.
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Adaptations and alterations of maternal microbiota: From physiology to pathology

TL;DR: A comprehensive understanding of maternal microbial adaptions and changes is provided, which brings insights into the association between maternal microbial dysbiosis and pregnancy complications and promotes the development of microbiota-specific approaches in the diagnosis and intervention of perinatal diseases.