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Nataliya L. Blatt

Researcher at Kazan Federal University

Publications -  21
Citations -  313

Nataliya L. Blatt is an academic researcher from Kazan Federal University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 6, co-authored 7 publications receiving 141 citations.

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World Health Organization Report: Current Crisis of Antibiotic Resistance

TL;DR: It sounds using the antibodies against problematic bacteria in farms has more benefits than treating them with susceptible antibiotics and the crisis of antibiotic resistance may be solved when all contributors be acknowledged to their responsibilities and duties to minimize this global problem threatening the human health.
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Genome-wide association analysis identifies genetic variations in subjects with myalgic encephalomyelitis/chronic fatigue syndrome

TL;DR: This study represents the most comprehensive genome-wide association study (GWAS) of an ME/CFS cohort conducted to date and identified 442 SNPs that cluster in the non-coding regions of T-cell receptor loci and may help identify contributing factors to the pathophysiology of ME/ CFS.
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Interaction and self-organization of human mesenchymal stem cells and neuro-blastoma SH-SY5Y cells under co-culture conditions: A novel system for modeling cancer cell micro-environment

TL;DR: The aim of this work was to study interactions between human mesenchymal stem cells and neuro-blastoma cancer SH-SY5Y cells under co-culture conditions on different coated surfaces to determine the effect of co-existence of cancer and stem cells on each cellular population under various stress conditions and to support the evidence that cancer andstem cell interactions play important roles in cellular behavior of cancer cells.
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Genetic Factors of Nitric Oxide's System in Psychoneurologic Disorders.

TL;DR: The contribution of genetic and environmental factors has been understudied, which makes this issue increasingly important for researchers as the understanding of these mechanisms can support a search for new approaches to pathogenetic and disease-modifying treatment.
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The Skin-Brain Connection Hypothesis, Bringing Together CCL27-Mediated T-Cell Activation in the Skin and Neural Cell Damage in the Adult Brain.

TL;DR: The observation of increased CC chemokine ligand 27 (CCL27) in MS sera supports the involvement of skin in the pathogenesis of MS and proposes that high serum levels of CCL27 in MS are the result of skin inflammation due to exposure to seasonal changes in the sunlight.