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Neil D. Woodward

Researcher at Vanderbilt University Medical Center

Publications -  121
Citations -  6245

Neil D. Woodward is an academic researcher from Vanderbilt University Medical Center. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 32, co-authored 111 publications receiving 5182 citations. Previous affiliations of Neil D. Woodward include University of Alberta & Alberta Hospital Edmonton.

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Dopaminergic Network Differences in Human Impulsivity

TL;DR: It is found that higher levels of trait impulsivity were predicted by diminished midbrain D2/D3 autoreceptor binding and greater amphetamine-induced DA release in the striatum, which was in turn associated with stimulant craving.
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A meta-analysis of neuropsychological change to clozapine, olanzapine, quetiapine, and risperidone in schizophrenia

TL;DR: The currently available evidence supporting cognitive improvement with atypical APDs was evaluated in two meta-analyses and revealed that atypicals are superior to typicals at improving overall cognitive function.
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Mesolimbic dopamine reward system hypersensitivity in individuals with psychopathic traits

TL;DR: These findings suggest that neurochemical and neurophysiological hyper-reactivity of the dopaminergic reward system may comprise a neural substrate for impulsive-antisocial behavior and substance abuse in psychopathy.
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Thalamocortical dysconnectivity in schizophrenia.

TL;DR: The etiology of schizophrenia may disrupt the development of prefrontal-thalamic connectivity and refinement of somatomotor connectivity with the thalamus that occurs during brain maturation, establishing differential abnormalities of thalamocortical networks in schizophrenia.
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Functional resting-state networks are differentially affected in schizophrenia

TL;DR: The results indicate that resting-state networks are differentially affected in schizophrenia, and the alterations are characterized by reduced segregation between the default mode and executive control networks in the prefrontal cortex and temporal lobe, and reduced connectivity in the dorsal attention andExecutive control networks.