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Nilson Sant'Anna

Researcher at National Institute for Space Research

Publications -  12
Citations -  378

Nilson Sant'Anna is an academic researcher from National Institute for Space Research. The author has contributed to research in topics: Total electron content & TEC. The author has an hindex of 7, co-authored 12 publications receiving 318 citations.

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An investigation of artificial neural networks based prediction systems in software project management

TL;DR: Artificial neural network and stepwise regression based predictive models are investigated, aiming at offering alternative methods for those who do not believe in estimation models and indicate that these techniques are competitive with the APF, SLIM, and COCOMO methods.
Proceedings ArticleDOI

Comparison of Artificial Neural Network and Regression Models in Software Effort Estimation

TL;DR: Predictive Artificial Neural Network and Regression based models are investigated, aiming at establishing simple estimation methods alternatives and show that artificial neural networks are effective in effort estimation.
Journal ArticleDOI

Ionospheric TEC Weather Map Over South America

TL;DR: Ionospheric weather maps using the total electron content (TEC) monitored by ground based GNSS receivers over South American continent, TECMAP, have been operationally produced by INPE´s Space Weather Study and Monitoring Program (Estudo e Monitoramento Brasileiro de Clima Especial, EMBRACE) since 2013.
Journal ArticleDOI

Plasma bubble monitoring by TEC map and 630 nm airglow image

TL;DR: In this paper, the total electron content (TECMAP) was used to monitor the equatorial ionosphere plasma bubbles over the South American continent by mapping the TECMAP using data provided by ground-based GNSS receiver networks.
Journal ArticleDOI

Antifibrotic Effects of Human Amniotic Membrane Transplantation in Established Biliary Fibrosis Induced in Rats.

TL;DR: The findings suggest that AM, when applied as a patch onto the liver surface, is useful for treating well-established cholestatic fibrosis, and the mechanism was partly by means of downregulating the profibrotic factor TGF-β1 and IL-6.