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Norbert Suttorp
Researcher at Humboldt State University
Publications - 143
Citations - 7668
Norbert Suttorp is an academic researcher from Humboldt State University. The author has contributed to research in topics: Endothelial stem cell & Endothelium. The author has an hindex of 48, co-authored 143 publications receiving 6883 citations. Previous affiliations of Norbert Suttorp include University of Giessen & Humboldt University of Berlin.
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Journal ArticleDOI
SARS-CoV-2-reactive T cells in healthy donors and patients with COVID-19.
Julian Braun,Lucie Loyal,Marco Frentsch,Daniel Wendisch,Philipp Georg,Florian Kurth,Florian Kurth,Stefan Hippenstiel,Manuela Dingeldey,Beate Kruse,Florent Fauchere,Emre Baysal,Maike Mangold,Larissa Henze,Roland Lauster,Roland Lauster,Marcus A. Mall,Marcus A. Mall,Kirsten Beyer,Jobst Röhmel,Sebastian Voigt,Jürgen Schmitz,Stefan Miltenyi,Ilja Demuth,Marcel A. Müller,Andreas C. Hocke,Martin Witzenrath,Norbert Suttorp,Florian Kern,Ulf Reimer,Holger Wenschuh,Christian Drosten,Victor M. Corman,Claudia Giesecke-Thiel,Leif E. Sander,Andreas Thiel +35 more
TL;DR: CD4 + T cells that are reactive against the spike glycoprotein of SARS-CoV-2 in the peripheral blood of patients with COVID-19 and healthy donors are found, indicating that spike-protein cross-reactive T cells are present and probably generated during previous encounters with endemic coronaviruses.
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CRB-65 predicts death from community-acquired pneumonia.
TL;DR: The study was performed to validate the CURB, CRB and CRB‐65 scores for the prediction of death from community‐acquired pneumonia (CAP) in both the hospital and out‐patient setting.
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Innate immune recognition in infectious and noninfectious diseases of the lung.
TL;DR: The PRR engagement is the prerequisite for the initiation of immune responses to infections and tissue injuries which can be beneficial or detrimental to the host.
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Nod1-Mediated Endothelial Cell Activation by Chlamydophila pneumoniae
Bastian Opitz,Stefanie Förster,Andreas C. Hocke,Matthias Maass,Bernd Schmeck,Stefan Hippenstiel,Norbert Suttorp,Matthias Krüll +7 more
TL;DR: In this paper, the authors examined the mechanisms of endothelial cell activation by C. pneumoniae via Nod proteins and found that Nod1 played a dominant role in triggering a chlamydia-mediated inflammatory process.
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Role of phosphodiesterases in the regulation of endothelial permeability in vitro.
TL;DR: The data suggest that adenylate cyclase activation/PDE inhibition is a powerful approach to block H2O2-induced increase in endothelial permeability, and appears especially valuable when endothelial PDE isoenzyme pattern and PDE inhibitor profile are matched optimally.