P
Paul M. Armistead
Researcher at University of North Carolina at Chapel Hill
Publications - 74
Citations - 1703
Paul M. Armistead is an academic researcher from University of North Carolina at Chapel Hill. The author has contributed to research in topics: Transplantation & Leukemia. The author has an hindex of 19, co-authored 64 publications receiving 1399 citations. Previous affiliations of Paul M. Armistead include Brigham and Women's Hospital & University of Texas MD Anderson Cancer Center.
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Journal ArticleDOI
Modification of Indium Tin Oxide Electrodes with Nucleic Acids: Detection of Attomole Quantities of Immobilized DNA by Electrocatalysis
TL;DR: Indium tin oxide electrodes were modified with DNA, and the guanines in the immobilized nucleic acid were used as a substrate for electrocatalytic oxidation by Ru(bpy)3(3+) (bpy = 2,2'-bipyridine), leading to adsorption of single-stranded DNA.
Journal ArticleDOI
Alternative tumour-specific antigens.
Christof C. Smith,Sara R. Selitsky,Shengjie Chai,Paul M. Armistead,Benjamin G. Vincent,Jonathan S. Serody +5 more
TL;DR: Examples of TSAs alternative to the traditional single-nucleotide variant neoantigens are provided and details about the novel computational tools used to identify them are provided, with the view to broaden the number of targetable antigens that can be used for cancer vaccine development.
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B cells from patients with chronic GVHD are activated and primed for survival via BAFF-mediated pathways.
Jessica L. Allen,Matthew S. Fore,Jenna Wooten,Philip A. Roehrs,Nazmim S. Bhuiya,Todd Hoffert,Andrew Sharf,Allison M. Deal,Paul M. Armistead,James M. Coghill,Don A. Gabriel,Robert Irons,Amber N Essenmacher,Thomas C. Shea,Kristy L. Richards,Corey Cutler,Jerome Ritz,Jonathan S. Serody,Albert S. Baldwin,Stefanie Sarantopoulos +19 more
TL;DR: A role for BAFF is identified in the pathogenesis of chronic GVHD and B-cell activation and survival pathways suitable for novel therapeutic development in cGVHD are defined.
Journal ArticleDOI
Oxidation kinetics of guanine in DNA molecules adsorbed onto indium tin oxide electrodes.
TL;DR: First- and second-order rate constants for catalytic and direct guanine oxidation were determined from the fast component and an increase in the local negative charge due to the oxygen sites on the ITO surface, and redox cycling of the catalyst, which maintains the surface concentration of the active form.
Journal ArticleDOI
Aberrant Mer receptor tyrosine kinase expression contributes to leukemogenesis in acute myeloid leukemia
Alisa B. Lee-Sherick,Kristen Eisenman,Susan Sather,A McGranahan,Paul M. Armistead,Colleen S. McGary,Sally A. Hunsucker,Jennifer Schlegel,Holly A. Martinson,C Cannon,Amy K. Keating,Henry Shelton Earp,Xiayuan Liang,Deborah DeRyckere,Doug Graham +14 more
TL;DR: A role for Mer in acute myeloid leukemogenesis is suggested and targeted inhibition of Mer may be an effective therapeutic strategy in pediatric and adult AML.