P
Peter A. Cattini
Researcher at University of Manitoba
Publications - 120
Citations - 3548
Peter A. Cattini is an academic researcher from University of Manitoba. The author has contributed to research in topics: Gene & Gene expression. The author has an hindex of 34, co-authored 119 publications receiving 3405 citations. Previous affiliations of Peter A. Cattini include St. Boniface General Hospital & University of California, San Francisco.
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Journal ArticleDOI
Biological activities of fibroblast growth factor-2 in the adult myocardium.
TL;DR: An overview of current insights into the multiple roles of FGF-2 in the myocardium, as they pertain to two basic phenomena: ischemia-reperfusion injury and cardiac hypertrophy, is given.
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Regulation of the higher-order structure of chromatin by histones H1 and H5.
TL;DR: Chicken erythrocyte chromatins containing a single species of linker histone, H1 or H5, have been prepared, using reassembly techniques developed previously, suggesting that the higher-order structure of chromatin is subject to secondary modulation by the associated histones.
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Phosphorylation of serine 262 in the gap junction protein connexin-43 regulates DNA synthesis in cell-cell contact forming cardiomyocytes.
Bradley W. Doble,Xitong Dang,Peipei Ping,Robert R. Fandrich,Barbara E. Nickel,Yan Jin,Peter A. Cattini,Elissavet Kardami +7 more
TL;DR: It is concluded that Cx43 inhibits cardiomyocyte DNA synthesis irrespectively of cell-cell contact or coupling and gap junction-mediated communication is required in order to reverse C x43 inhibition of DNA synthesis by S262 phosphorylation.
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High and Low Molecular Weight Fibroblast Growth Factor-2 Increase Proliferation of Neonatal Rat Cardiac Myocytes but Have Differential Effects on Binucleation and Nuclear Morphology: Evidence for Both Paracrine and Intracrine Actions of Fibroblast Growth Factor-2
TL;DR: Results suggest that 22/21.5- kD FGF-2 and 18-kD F GF-2 have similar paracrine effects on proliferation but that 22-21.
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HNF-1α G319S, a transactivation-deficient mutant, is associated with altered dynamics of diabetes onset in an Oji-Cree community
Barbara Triggs-Raine,Robert D. Kirkpatrick,Sherrie L Kelly,Lisa D. Norquay,Peter A. Cattini,Kazuya Yamagata,Anthony J. Hanley,Bernard Zinman,Stewart B. Harris,P. Hugh R. Barrett,Robert A. Hegele +10 more
TL;DR: The demonstration of a functional consequence for HNF1A G319S provides a mechanistic basis for its strong association with Oji-Cree type 2 diabetes and its unparalleled specificity for diabetes prediction in these people, in whom diabetes presents a significant public health dilemma.