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Peter Ochodnicky

Researcher at University of Amsterdam

Publications -  28
Citations -  847

Peter Ochodnicky is an academic researcher from University of Amsterdam. The author has contributed to research in topics: Kidney & Kidney disease. The author has an hindex of 16, co-authored 28 publications receiving 761 citations. Previous affiliations of Peter Ochodnicky include University Medical Center Groningen & Comenius University in Bratislava.

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Journal ArticleDOI

Microalbuminuria and endothelial dysfunction: emerging targets for primary prevention of end-organ damage.

Peter Ochodnicky, +3 more
- 01 Jan 2006 - 
TL;DR: It is proposed that microalbuminuria and endothelial dysfunction are an emerging target for primary prevention strategies in cardiovascular disease and dietary components improving nitric oxide bioavailability, such as cocoa-derived flavanols may play important role in these preventive strategies.
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Perinatally administered losartan augments renal ACE2 expression but not cardiac or renal Mas receptor in spontaneously hypertensive rats

Jan Klimas, +7 more
- 01 Aug 2015 - 
TL;DR: Prevention of hypertension and LV hypertrophy development by losartan was unrelated to cardiac or renal expression of Mas, and the influence of locally generated Ang‐(1‐7) in organ response to the developing hypertension in SHRs is suggested.
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Isoproterenol‐induced heart failure in the rat is associated with nitric oxide‐dependent functional alterations of cardiac function

Peter Krenek, +8 more
- 01 Feb 2009 - 
TL;DR: The hypothesis that the role of NO in isolated atria and cardiomyocytes is altered in isoproterenol‐induced HF is tested.
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Neurotrophins as regulators of urinary bladder function

Peter Ochodnicky, +3 more
- 01 Nov 2012 - 
TL;DR: Accumulating evidence points towards a role for neurotrophins in the control of neural sensory function during micturition and indicates their involvement in the emergence of OAB-related and BPS-related LUTS.
Journal Article

Endothelial dysfunction in chronic kidney disease: determinant of susceptibility to end-organ damage and therapeutic response.

Peter Ochodnicky, +5 more
- 01 May 2006 - 
TL;DR: Endothelial dysfunction seems to be a crucial mediator of increased cardiovascular risk observed among patients with chronic kidney disease, and may represent an ideal therapeutic target not only for treatment, but also for primary prevention of renal disease.