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Philip O. Ettinger
Researcher at Rutgers University
Publications - 25
Citations - 1678
Philip O. Ettinger is an academic researcher from Rutgers University. The author has contributed to research in topics: Cardiac output & Alcoholic cardiomyopathy. The author has an hindex of 14, co-authored 25 publications receiving 1623 citations.
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Journal ArticleDOI
Arrhythmias and the “Holiday Heart”: Alcoholassociated cardiac rhythm disorders
Philip O. Ettinger,Chia F. Wu,Catalino De La Cruz,Allen B. Weisse,S. Sultan Ahmed,Timothy J. Regan +5 more
TL;DR: Cardiac arrhythmias presenting during weekend or holiday drinking episodes are associated with conduction delays and depressed cardiac performance indicative of early cardiomyopathy and suggest a “holiday heart” syndrome.
Journal ArticleDOI
Altered Myocardial Function and Metabolism in Chronic Diabetes Mellitus without Ischemia in Dogs
Timothy J. Regan,Philip O. Ettinger,Mohammad I. Khan,Mohan U. Jesrani,Michael M. Lyons,Henry A. Oldewurtel,Marilyn Weber +6 more
TL;DR: It was concluded that chronic diabetes mellitus can alter myocardial composition and function independent of vascular effects.
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Systolic time intervals as measures of the contractile state of the left ventricular myocardium in man.
TL;DR: The results indicate that the systolic times are a valid measure of contractility which should prove useful in comparing patients with cardiac pathology confined to the LV myocardium and in following patients with extramyocardial hemodynamic lesions of constant severity.
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Hyperkalemia, cardiac conduction, and the electrocardiogram: A review
TL;DR: Although slow systemic increase of K + induces asystole in animals, administration to a region of myocardium produces ventricular ectopic beats and fibrillation, the latter effect may be due to re-entry and may be mechanistically similar to the arrhythmias of early ischemia.
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Cardiac conduction abnormalities produced by chronic alcoholism.
TL;DR: Extended ethanol intake in the absence of evident malnutrition resulted in demonstrable intraventricular conduction abnormalities and morphologic alterations which were related to duration of ingestion, consistent with a cumulative toxic effect of ethanol.