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Philip P.C. Ip

Researcher at University of Hong Kong

Publications -  95
Citations -  2981

Philip P.C. Ip is an academic researcher from University of Hong Kong. The author has contributed to research in topics: Cancer & Carcinoma. The author has an hindex of 26, co-authored 86 publications receiving 2460 citations. Previous affiliations of Philip P.C. Ip include Zhengzhou University & Queen Mary University of London.

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HBsAg Seroclearance in Chronic Hepatitis B in Asian Patients: Replicative Level and Risk of Hepatocellular Carcinoma

TL;DR: HBsAg seroclearance at age <50 years was associated with a lower risk for the development of HCC, and HBV persisted at low replicative and transcriptional levels after HBsAgSerolearance.
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Aberrant activation of hedgehog signaling pathway in ovarian cancers: effect on prognosis, cell invasion and differentiation

TL;DR: The data suggested that abnormal HH signaling activation plays important roles in the development and progression of ovarian cancers and inhibition of the HH pathway molecules might be a valid therapeutic strategy for ovarian cancers.
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Uterine smooth muscle tumors of uncertain malignant potential (STUMP): a clinicopathologic analysis of 16 cases

TL;DR: Uterine tumors classified as STUMPs using criteria proposed by Stanford investigators are usually clinically benign but should be considered tumors of low malignant potential because they can occasionally recur, in some cases, years after hysterectomy.
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Uterine smooth muscle tumors other than the ordinary leiomyomas and leiomyosarcomas: a review of selected variants with emphasis on recent advances and unusual morphology that may cause concern for malignancy.

TL;DR: The current review discusses the pathologic diagnosis of and terminology applied to selected variants of uterine smooth muscle tumors other than the ordinary leiomyomas and leoomyosarcomas with emphasis on unusual reported features that may indicate malignancy.
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Soluble E-cadherin promotes tumor angiogenesis and localizes to exosome surface.

TL;DR: It is shown for the first time that soluble E-cadherin is released by ovarian cancer cells packaged in exosomes and promotes tumor angiogenesis through β-catenin and NFkB signaling activation.