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Pulkit Grover

Researcher at Carnegie Mellon University

Publications -  191
Citations -  5602

Pulkit Grover is an academic researcher from Carnegie Mellon University. The author has contributed to research in topics: Decoding methods & Counterexample. The author has an hindex of 27, co-authored 176 publications receiving 4874 citations. Previous affiliations of Pulkit Grover include Stanford University & University of California, Berkeley.

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CodeNet: Training Large Scale Neural Networks in Presence of Soft-Errors.

TL;DR: The experiments show that CodeNet achieves the best accuracy-runtime tradeoff compared to both replication and uncoded strategies, and is a significant step towards biologically plausible neural network training, that could hold the key to orders of magnitude efficiency improvements.
Journal ArticleDOI

Information Embedding and the Triple Role of Control

TL;DR: The results provide the tightest known lower bounds on the asymptotic costs for the vector version of a famous open problem in decentralized control: the Witsenhausen counterexample.
Proceedings ArticleDOI

How far are LDPC codes from fundamental limits on total power consumption

TL;DR: How close LDPC codes with corresponding message-passing decoders get to the fundamental limits is investigated, finding that the transmit power needs to increase unboundedly in order for the total power to be asymptotically smaller than that for uncoded transmission.
Posted ContentDOI

Very high density EEG elucidates spatiotemporal aspects of early visual processing

TL;DR: Overall, SND EEG captured more neural information from visual cortex, arguing for increased development of this approach in basic and translational neuroscience.
Proceedings ArticleDOI

Do single neuron models exhibit temporal interference stimulation

TL;DR: It is demonstrated that the classical Hodgkin-Huxley squid neurons and some mammalian cells do exhibit TI stimulation in this model, and deeper studies are needed to understand if TI stimulation works with all neuron-types, or if it is network mechanisms (instead of single neuron dynamics) that enable TI stimulation.