Q
Qing Zhao
Researcher at Lund University
Publications - 20
Citations - 1080
Qing Zhao is an academic researcher from Lund University. The author has contributed to research in topics: Sciatic nerve & Reinnervation. The author has an hindex of 15, co-authored 20 publications receiving 1059 citations. Previous affiliations of Qing Zhao include Malmö University & Rush University Medical Center.
Papers
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Journal ArticleDOI
Can sensory and motor collateral sprouting be induced from intact peripheral nerve by end-to-side anastomosis?
G. Lundborg,Qing Zhao,Qing Zhao,M Kanje,M Kanje,Nils Danielsen,Nils Danielsen,James M. Kerns,James M. Kerns +8 more
TL;DR: The findings suggest that collateral sprouting may occur from intact axons, perhaps induced by factors emanating from the attached nerve segment, and subsequently make functional peripheral connections.
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Trophism, tropism, and specificity in nerve regeneration.
TL;DR: The capacity of the brain to "reprogram" itself and adapt to this functional re-organization is critical for the ultimate recovery of functional sensory/motor function after nerve injuries.
Journal ArticleDOI
Pituitary adenylate cyclase activating peptide expression in the rat dorsal root ganglia : up-regulation after peripheral nerve injury
TL;DR: The data obtained indicate a marked up-regulation of PACAP in sensory neurons following peripheral nerve injury, which may have implications for sensory transmission.
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Rat sciatic nerve regeneration through a micromachined silicon chip
Qing Zhao,Johan Drott,Thomas Laurell,Lars Wallman,Kjell Lindström,Lars Magnus Bjursten,Göran Lundborg,Lars Montelius,Nils Danielsen +8 more
TL;DR: The capacity of regenerating nerve fibres to grow through a perforated silicon chip was tested using the silicone chamber model for nerve regeneration and the muscle contractility force of the gastrocnemius muscle was measured after 16 weeks of regeneration and compared as a percentage of the contralateral uninjured side.
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Pre-degenerated nerve grafts enhance regeneration by shortening the initial delay period.
TL;DR: The results suggest that the effects of pre-degeneration are not only due to the increased cell proliferation in the graft, but that also trophic and/or inflammatory mechanisms may be of importance.