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Ramona Lall

Researcher at New York University

Publications -  23
Citations -  1964

Ramona Lall is an academic researcher from New York University. The author has contributed to research in topics: Environmental exposure & Air pollution. The author has an hindex of 14, co-authored 21 publications receiving 1755 citations.

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Journal ArticleDOI

Ischemic Heart Disease Mortality and Long-Term Exposure to Source-Related Components of U.S. Fine Particle Air Pollution

TL;DR: It is suggested that PM2.5–mortality associations can vary greatly by source, and that the largest IHD health benefits per microgram/cubic meter from PM 2.5 air pollution control may be achieved via reductions of fossil fuel combustion exposures, especially from coal-burning sources.
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PM source apportionment and health effects: 1. Intercomparison of source apportionment results.

TL;DR: Overall, although these intercomparisons suggest areas where further research is needed, they provide support the contention that PM2.5 mass source apportionment results are consistent across users and methods, and that today's sourceapportionment methods are robust enough for application to PM2-5 health effects assessments.
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A source apportionment of U.S. fine particulate matter air pollution

TL;DR: The results indicate that applying source apportionment methods to the nationwide CSN can be an informative avenue for identifying and quantifying source components for the subsequent estimation of source-specific health effects, potentially contributing to more efficient regulation of PM2.5.
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Time-series analysis of mortality effects of fine particulate matter components in Detroit and Seattle.

TL;DR: The effects of PM2.5 on daily mortality vary with source, season, and locale, consistent with the hypothesis that PM composition has an appreciable influence on the health effects attributable to PM.
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Personal exposures to traffic-related air pollution and acute respiratory health among Bronx schoolchildren with asthma.

TL;DR: Adverse health associations were strongest with personal measures of EC exposure, suggesting that the diesel “soot” fraction of PM2.5 is most responsible for pollution-related asthma exacerbations among children living near roadways.