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Reynaldo D. Pinto

Researcher at University of São Paulo

Publications -  117
Citations -  1651

Reynaldo D. Pinto is an academic researcher from University of São Paulo. The author has contributed to research in topics: Thin film & Pulsed laser deposition. The author has an hindex of 18, co-authored 117 publications receiving 1588 citations. Previous affiliations of Reynaldo D. Pinto include Tata Institute of Fundamental Research & University of California, San Diego.

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Synchronous behavior of two coupled electronic neurons

TL;DR: In this paper, a pair of analog electronic neurons (ENs) were designed to reproduce the observed membrane voltage oscillations of isolated biological neurons from the stomatogastric ganglion of the California spiny lobster Panulirus interruptus.
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Size effect study in magnetoelectric BiFeO3 system

TL;DR: In this article, finite size effects on Neel temperature (T N) of magnetoelectric BiFeO3 system were reported for the first time, and the decrease in T N was co-related to unit cell volume contraction occurring with reduction in particle size.
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Spin-polarized tunneling in the half-metallic ferromagnets La 0.7-x Ho x Sr 0.3 MnO 3 (x=0 and 0.15): Experiment and theory

TL;DR: In this paper, the authors derived a theoretical expression for the contribution of spin-polarized tunneling to the magnetoresistance in granular ferromagnetic systems under the mean-field approximation.
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Extended dynamic clamp: controlling up to four neurons using a single desktop computer and interface

TL;DR: This work describes how to extend dynamic clamp control to four neurons and their associated synaptic interactions, using a single IBM-compatible PC, an ADC/DAC interface with two analog outputs, and an additional demultiplexing circuit.
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Reliable circuits from irregular neurons: A dynamical approach to understanding central pattern generators

TL;DR: The Hindmarsh-Rose model was used as the basis for building electronic neurons which could then be integrated into the biological circuitry and were able to rescue patterns which had been disabled by removing key biological neurons from the circuit.