R
Riccardo Dalla-Favera
Researcher at New York University
Publications - 58
Citations - 5147
Riccardo Dalla-Favera is an academic researcher from New York University. The author has contributed to research in topics: Lymphoma & Gene. The author has an hindex of 33, co-authored 58 publications receiving 5115 citations. Previous affiliations of Riccardo Dalla-Favera include Harvard University & Columbia University.
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Journal ArticleDOI
Expression of cellular homologues of retroviral onc genes in human hematopoietic cells
Eric H. Westin,Flossie Wong-Staal,Edward P. Gelmann,Riccardo Dalla-Favera,Takis S. Papas,James A. Lautenberger,Alessandra Eva,E P Reddy,Steven R. Tronick,Stuart A. Aaronson,Robert C. Gallo +10 more
TL;DR: Total cellular poly(A)-enriched RNA from a variety of fresh human leukemic blood cells and hematopoietic cell lines was analyzed for homology with molecularly cloned DNA probes containing the onc sequence of Abelson murine leukemia virus (Ab-MuLV), Harvey murine sarcoma virus (Ha-MuSV), simian sarcomA virus (SSV), and avian myelocytomatosis virus strain MC29.
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Lymphoid Neoplasia Associated with the Acquired Immunodeficiency Syndrome (AIDS): The New York University Medical Center Experience with 105 Patients (1981-1986)
Daniel M. Knowles,Glenn A. Chamulak,Milayna Subar,Jerome S. Burke,Margaret Dugan,J C Wernz,Chrystia M. Slywotzky,Pier Giuseppe Pelicci,Riccardo Dalla-Favera,Bruce Raphael +9 more
TL;DR: This article identified 105 patients with lymphoid neoplasia associated with the acquired immunodeficiency syndrome (AIDS) at the New York University Medical Center from 1981 through 1986: 89 had non-Hodgkin lymphoma; 13, Hodgkin disease; and 3, chronic lymphocytic leukemia.
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Chromosomal breakpoints and structural alterations of the c-myc locus differ in endemic and sporadic forms of Burkitt lymphoma
TL;DR: Different genetic mechanisms may be involved in chromosomal translocation/c-myc activation, and these differences may be a function of differences in the stage of differentiation of eBL versus sBL.
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Cloning and characterization of different human sequences related to the onc gene (v-myc) of avian myelocytomatosis virus (MC29).
Riccardo Dalla-Favera,Edward P. Gelmann,Stefano Martinotti,Genoveffa Franchini,Takis S. Papas,Robert C. Gallo,Flossie Wong-Staal +6 more
TL;DR: Restriction mapping experiments and heteroduplex analysis show that c-myc sequences of this locus are interrupted by one intron, suggesting that lambda-LMC-12 and -41 contain the complete functional c- myc gene.
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Pathogenesis of Burkitt lymphoma: Expression of an activated c-myc oncogene causes the tumorigenic conversion of EBV-infected human B lymphoblasts
TL;DR: EBV infection and c-myc activation are sufficient for the tumorigenic conversion of human B cells in vitro, strongly supporting the hypothesis that these same two pathogenetic steps may be involved in the in vivo development of Burkitt lymphoma.