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Richard D. Todd

Researcher at Washington University in St. Louis

Publications -  174
Citations -  18738

Richard D. Todd is an academic researcher from Washington University in St. Louis. The author has contributed to research in topics: Population & Attention deficit hyperactivity disorder. The author has an hindex of 70, co-authored 174 publications receiving 17845 citations. Previous affiliations of Richard D. Todd include University of Missouri–St. Louis.

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Subgenual prefrontal cortex abnormalities in mood disorders

TL;DR: Using positron emission tomographic images of cerebral blood flow and rate of glucose metabolism to measure brain activity, an area of abnormally decreased activity is localized in the pre-frontal cortex ventral to the genu of the corpus callosum in both familial bipolar depressives and familial unipolar depressives.
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Validation of a brief quantitative measure of autistic traits: comparison of the social responsiveness scale with the autism diagnostic interview-revised.

TL;DR: The Social Responsiveness Scale is a valid quantitative measure of autistic traits, feasible for use in clinical settings and for large-scale research studies of autism spectrum conditions.
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Autistic traits in the general population: a twin study.

TL;DR: The data indicate that the social deficits characteristic of autism spectrum disorders are common, and it may be arbitrary where cutoffs are made between research designations of being "affected" vs "unaffected" with a pervasive developmental disorder.
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Intergenerational Transmission of Subthreshold Autistic Traits in the General Population

TL;DR: Children from families in which both parents manifest subthreshold autistic traits exhibit a substantial shift in the distribution of their scores for impairment in reciprocal social behavior, toward the pathological end.
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Glucose metabolism in the amygdala in depression: relationship to diagnostic subtype and plasma cortisol levels.

TL;DR: Data confirm the previous finding that neurophysiological activity is abnormally increased in FPDD, and extend it to BD-D, as well as confirming the correlation between amygdala metabolism and stressed plasma cortisol levels.