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Robert Anton

Researcher at Centre national de la recherche scientifique

Publications -  99
Citations -  3531

Robert Anton is an academic researcher from Centre national de la recherche scientifique. The author has contributed to research in topics: Psychotria forsteriana & Ginkgo biloba. The author has an hindex of 31, co-authored 99 publications receiving 3341 citations. Previous affiliations of Robert Anton include University of Valle & University of Strasbourg.

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Natural Dietary Polyphenolic Compounds Cause Endothelium-Dependent Vasorelaxation in Rat Thoracic Aorta

TL;DR: Results show that anthocyanins and oligomeric-condensed tannins exhibited a pharmacological profile comparable to the original RWPC, which may be involved in the reduction of cardiovascular mortality related to the presence of wine, fruits and vegetables in the diet.
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Antiprotozoal activities of Colombian plants.

TL;DR: The methylene chloride extract of leaves from C. scoparioides showed a selectivity index in the same range that the one of the Glucantime control, and several of the active leishmanicidal plants are traditionally used against leish maniasis by the population of the concerned area.
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Superoxide anion scavenging effect and superoxide dismutase activity ofGinkgo biloba extract

TL;DR: This work studies the action of Gbe against superoxide anion, which is directly or indirectly implicated in cell damage, and appears to have both an scavenging effect and also a superoxide dismutase activity.
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Inhibition of aggregation and secretion of human platelets by quercetin and other flavonoids: structure-activity relationships.

TL;DR: Quercetin and 12 other natural flavonoid aglycones inhibit washed human platelet aggregation and secretion of serotonin induced by ADP, collagen or thrombin.
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Cytotoxic activity of Amaryllidaceae alkaloids.

TL;DR: Almost all of the tested alkaloids showed cytotoxic activity against fibroblastic LMTK cells, and mesembrenone showed some specificity against Molt4 cells in comparison to LMTk cells, while lycorenine was found to be the most cytot toxic compound against HepG2 hepatoma.