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Showing papers by "Robert V. Kenyon published in 1984"


Journal ArticleDOI
TL;DR: Saccadic eye movements from three patients diagnosed as having Korsakoff's syndrome and one patient who had suffered the acute Wernicke stage of the disorder were examined for changes in saccadic characteristics.
Abstract: Saccadic eye movements from three patients diagnosed as having Korsakoff's syndrome and one patient who had suffered the acute Wernicke stage of the disorder were examined for changes in saccadic characteristics. The three Korsakoff patients showed increased saccadic latencies; two of the three also had reduced saccadic peak velocities and increased saccadic durations. A higher than expected incidence of hypometric saccades was also found in all four patients; saccadic intrusions were recorded in two Korsakoff patients. These abnormal oculomotor responses were found 2-7 years after the onset of the Korsakoff's syndrome and are consistent with the cerebellar and frontal lobe dysfunction reported in this patient group.

30 citations


Journal ArticleDOI
TL;DR: This work presents objective, quantitative, static and dynamic records of accommodation, vergence and related system responses in a symptomatic patient having accommodation disfacility and intermittent exotropia and proposes that these two conditions worked in a pathogenic symbiotic manner.

11 citations


Journal ArticleDOI
TL;DR: A severe disturbance in smooth pursuit function is demonstrated long after the clinically apparent oculomotor abnormalities have passed in patients with alcoholic Korsakoff's syndrome and an age-matched control.
Abstract: Smooth pursuit eye movements were studied in three patients with alcoholic Korsakoff's syndrome, one with Wernicke's encephalopathy, and an age-matched control. Horizontal smooth pursuit eye movements were abnormal in all patients: peak eye velocity and the ability to sustain smooth eye velocity were reduced. Also, smooth pursuit gain began to decrease at relatively low target velocities (i.e., 8–10°). These data demonstrate a severe disturbance in smooth pursuit function long after the clinically apparent oculomotor abnormalities have passed.

7 citations