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Robert W. Schrier

Researcher at University of Colorado Denver

Publications -  595
Citations -  41275

Robert W. Schrier is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Vasopressin & Renal function. The author has an hindex of 99, co-authored 586 publications receiving 38996 citations. Previous affiliations of Robert W. Schrier include Parke-Davis & University of Texas Health Science Center at San Antonio.

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Journal ArticleDOI

The acute effect of chlorothiazide on serum-ionized calcium. Evidence for a parathyroid hormone-dependent mechanism.

Mordecai M. Popovtzer, +4 more
- 01 Jun 1975 - 
TL;DR: In normal subjects and those with hypoparathyroidism, CTZ plus parathyroid extract infusion resulted in sustained increases in both SCA-plus 2 and TSCA throughout the periods of observation when compared to experiments in which only parathyro extract was infused, P smaller than 0.01 in all instances.
Journal ArticleDOI

Renal ultrasonographic evaluation in children at risk of autosomal dominant polycystic kidney disease.

Berenice Reed, +8 more
- 01 Jul 2010 - 
TL;DR: The present results in 420 at-risk children with ADPKD 15 years or younger detected bilateral renal cysts using ultrasonography in 181 of the children who had a family history of this genetic disease.
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Identification and characterization of a novel hypertonicity-responsive element in the human aquaporin-1 gene.

Fuminori Umenishi, +1 more
- 05 Apr 2002 - 
TL;DR: The data suggest that the transcription of the AQP1 by hypertonicity in renal cells is upregulated by the interaction with putative DNA binding proteins to a novel HRE located at -54 to -46 in the AQp1 gene.
Journal Article

Time-dependent protective effects of calcium channel blockers on anoxia- and hypoxia-induced proximal tubule injury.

A. R. P. Almeida, +5 more
- 01 Feb 1992 - 
TL;DR: Early membrane injury to isolated rPT in suspension, which is associated with 10 min of either anoxia or hypoxia, involves increased cellular Ca++ uptake through voltage-sensitive Ca++ channels and protection is afforded by CCB.
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Effect on stability, degradation, expression, and targeting of aquaporin-2 water channel by hyperosmolality in renal epithelial cells

Fuminori Umenishi, +2 more
- 23 Dec 2005 - 
TL;DR: Results indicate that hyperosmolality plays an important role in the stability, degradation, expression, and targeting of ng-AQP2 and that urea enhanced it compared to isotonic conditions.