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Roger Williams

Researcher at University of Cambridge

Publications -  1468
Citations -  74266

Roger Williams is an academic researcher from University of Cambridge. The author has contributed to research in topics: Hepatitis & Liver transplantation. The author has an hindex of 122, co-authored 1455 publications receiving 72416 citations. Previous affiliations of Roger Williams include Armed Forces Institute of Pathology & St Bartholomew's Hospital.

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Transection of the oesophagus for bleeding oesophageal varices

TL;DR: Emergency ligation of bleeding oesophageal varices using the Milnes Walker technique was performed in 38 patients, and in patients with good preoperative liver function this rose to 71% and the simple scoring system for grading the severity of disturbance of liver function was found to be of value in predicting the outcome of surgery.
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Long-Term Outcome of Hepatitis C Infection after Liver Transplantation

TL;DR: After liver transplantation for HCV-related cirrhosis, persistent HCV infection can cause severe graft damage, and such damage is more frequent in patients infected with HCV genotype 1b than with other genotypes.
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Acute liver failure: redefining the syndromes

TL;DR: Hyperacute liver failure is the authors' suggested term for cases in which encephalopathy occurs within 7 days of the onset of jaundice; this group includes the sizeable cohort likely to survive with medical management despite the high incidence of cerebral oedema.
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Global challenges in liver disease

Roger Williams
- 01 Sep 2006 - 
TL;DR: Only through a concerted effort from governments, health agencies, healthcare professionals at all levels, and the pharmaceutical industry can this grim outlook for liver disease worldwide be reversed.
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Influence of delta infection on severity of hepatitis b

TL;DR: In 25 of the 43 patients with delta-positive fulminant hepatitis, serum markers indicated a primary hepatitis B infection while in the remaining 18, IgM antibody to hepatitis B core antigen was absent, indicating that hepatitis B preceded superinfection with the delta agent.