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Rolf Lood

Researcher at Lund University

Publications -  44
Citations -  1146

Rolf Lood is an academic researcher from Lund University. The author has contributed to research in topics: Propionibacterium acnes & Bacteriophage. The author has an hindex of 14, co-authored 40 publications receiving 833 citations. Previous affiliations of Rolf Lood include Rockefeller University.

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Biofilm formation by Propionibacterium acnes is a characteristic of invasive isolates

TL;DR: The presence of human plasma in solution or at the plastic surface inhibits biofilm formation, which could explain why P. acnes primarily infect plasma-poor environments of, for example, joint prostheses and cerebrospinal shunts.
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Novel Phage Lysin Capable of Killing the Multidrug-Resistant Gram-Negative Bacterium Acinetobacter baumannii in a Mouse Bacteremia Model

TL;DR: PlyF307 represents the first highly active therapeutic lysin specific for Gram-negative organisms in an array of native lysins found in Acinetobacter phage and rescued mice from lethal A. baumannii bacteremia.
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Novel Engineered Peptides of a Phage Lysin as Effective Antimicrobials against Multidrug-Resistant Acinetobacter baumannii.

TL;DR: The prospect of using peptide derivatives from bacteriophage lysins to treat topical infections and remove biofilms caused by Gram-negative pathogens is demonstrated.
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Revisiting Antibiotic Resistance Spreading in Wastewater Treatment Plants - Bacteriophages as a Much Neglected Potential Transmission Vehicle.

TL;DR: Current literature in the role of WWTPs as reservoirs and hotspots of antibiotic resistance with a specific focus on bacteriophages as mediators of genetic exchange is examined, highlighting the importance of transduction in WWTBs as a mediator of resistance spread.
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IgG glycan hydrolysis by endoglycosidase S diminishes the proinflammatory properties of immune complexes from patients with systemic lupus erythematosus: A possible new treatment?

TL;DR: Findings indicate that endoglycosidase S treatment has prominent effects on several pathogenetically important IC-mediated events, and suggest that EndoS has the potential to be developed as a novel therapy for SLE.