R
Ronald A. Morton
Researcher at Baylor College of Medicine
Publications - 45
Citations - 4338
Ronald A. Morton is an academic researcher from Baylor College of Medicine. The author has contributed to research in topics: Prostate cancer & Prostate. The author has an hindex of 26, co-authored 45 publications receiving 4248 citations. Previous affiliations of Ronald A. Morton include Johns Hopkins University & Johns Hopkins University School of Medicine.
Papers
More filters
Journal ArticleDOI
Cytidine methylation of regulatory sequences near the pi-class glutathione S-transferase gene accompanies human prostatic carcinogenesis
Wen-Hsiang Lee,Ronald A. Morton,Jonathan I. Epstein,James D. Brooks,Pearl A. Campbell,G S Bova,Wen Son Hsieh,William B. Isaacs,William G. Nelson +8 more
TL;DR: Methylation of cytidine nucleotides in GSTP1 regulatory sequences constitutes the most common genomic alteration yet described for human prostate cancer.
Journal Article
Metastatic Prostate Cancer in a Transgenic Mouse
Jeffrey R. Gingrich,Roberto Barrios,Ronald A. Morton,Brendan F. Boyce,Francesco J. DeMayo,Milton J. Finegold,Roxani Angelopoulou,Jeffrey M. Rosen,Norman M. Greenberg +8 more
TL;DR: The TRAMP model provides a consistent source of primary and metastatic tumors for histopathobiological and molecular analysis to further define the earliest molecular events involved in the genesis, progression, and metastasis of prostate cancer.
Journal ArticleDOI
Impact of Anatomical Radical Prostatectomy on Urinary Continence
TL;DR: It is suggested that anatomical factors rather than preservation of autonomic innervation may be responsible for the improved urinary control associated with an anatomical approach to radical prostatectomy.
Journal ArticleDOI
Radical retropubic prostatectomy. Improved anastomosis and urinary continence.
TL;DR: Identification of the striated urethral sphincter ranks in importance with elucidation of the anatomy of the dorsal vein complex and the anatomic identification of the neurovascular bundle in improving the patient's quality of life postoperatively.
Journal Article
Reduction of E-Cadherin Levels and Deletion of the α-Catenin Gene in Human Prostate Cancer Cells
Ronald A. Morton,Charles M. Ewing,Charles M. Ewing,Akira Nagafuchi,Shoichiro Tsukita,William B. Isaacs +5 more
TL;DR: It is confirmed that E-cadherin-dependent cell-cell adhesion is frequently aberrant in prostate cancer cells, and suggested that in a subset of prostate cancers, this adhesion may be inactivated by loss of α-catenin rather than E- cadher in itself.