Rosaria Plasmati

TL;DR: TMS, as currently used for monitoring conduction in central motor pathways, does not induce seizures in drug-treated epileptic patients.

TL;DR: VBM, histogram and TBSS results correlated with clinical severity of clinical deficit in SCA1 and SCA2 and brain WM damage featuring a pontocerebeellar atrophy in both diseases correlates with severity of the clinical deficit.

TL;DR: The restless legs syndrome responded favourably to clonazepam and was the first isolated clinical manifestation in four siblings of a family with familial amyloid polyneuropathy.

TL;DR: Minor decreases in the N‐acetylaspartate/Creatine ratio and the normal choline/creatine ratio, demonstrated by MR spectroscopy in the pons, is likely to reflect a loss of neuronal viability and might represent a biochemical marker of SCA1 more sensitive than brainstem and cerebellum atrophy and signal changes shown by MR imaging.

TL;DR: Transthyretin-related amyloidsosis may cause superficial siderosis of the CNS through subarachnoid bleeding related to meningovascular amyloid deposition.