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Ruth I. Wood

Researcher at University of Southern California

Publications -  93
Citations -  4509

Ruth I. Wood is an academic researcher from University of Southern California. The author has contributed to research in topics: Androgen & Testosterone (patch). The author has an hindex of 35, co-authored 92 publications receiving 4131 citations. Previous affiliations of Ruth I. Wood include Yale University.

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Adverse Health Consequences of Performance-Enhancing Drugs: An Endocrine Society Scientific Statement

TL;DR: This scientific statement synthesizes available information on the medical consequences of P ED use, identifies gaps in knowledge, and aims to focus the attention of the medical community and policymakers on PED use as an important public health problem.
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Targeted disruption of the pituitary glycoprotein hormone alpha-subunit produces hypogonadal and hypothyroid mice.

TL;DR: The pituitary cells that produce TSH beta-subunit exhibited dramatic hypertrophy and hyperplasia as a result of the lack of thyroid function, consistent with a derivation of these three cell types from a common precursor.
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Anabolic–androgenic steroid dependence: an emerging disorder

TL;DR: The accumulating human and animal evidence showing that AAS may cause a distinct dependence syndrome is reviewed, and standard diagnostic criteria for substance dependence must be adapted slightly for cumulatively acting drugs such as AAS.
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Bidirectional connections of the medial amygdaloid nucleus in the Syrian hamster brain: simultaneous anterograde and retrograde tract tracing.

TL;DR: Combined anterograde and retrograde tract tracers demonstrate that subnuclei of Me are interconnected with limbic structures in hamster brain, which may contribute to chemosensory and hormonal integration to control male sexual behavior.
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Metabolic interfaces between growth and reproduction. I. Nutritional modulation of gonadotropin, prolactin, and growth hormone secretion in the growth-limited female lamb.

TL;DR: Dietary restriction in the developing female lamb depresses gonadotropin secretion without reducing other anterior pituitary gland secretions, such as PRL and GH, which implies that metabolic and growth-related modulation of neuroendocrine function can occur independently of changes in sensitivity to the feedback actions of ovarian steroids and polypeptides.