S
Sam Kacew
Researcher at University of Ottawa
Publications - 149
Citations - 4166
Sam Kacew is an academic researcher from University of Ottawa. The author has contributed to research in topics: Kidney & Chlorphentermine. The author has an hindex of 31, co-authored 146 publications receiving 3680 citations. Previous affiliations of Sam Kacew include Sungkyunkwan University.
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Journal ArticleDOI
Human Health Risk Assessment for Aluminium, Aluminium Oxide, and Aluminium Hydroxide
Daniel Krewski,Robert A. Yokel,Evert Nieboer,David R. Borchelt,Joshua T. Cohen,Jean Harry,Sam Kacew,Joan Lindsay,Amal M Mahfouz,Virginie Rondeau +9 more
TL;DR: This article was originally published with an incorrect version of the Acknowledgments, which appeared on p. 218 of the print version.
Journal ArticleDOI
Drug-induced phospholipidosis: are there functional consequences?
Mark J. Reasor,Sam Kacew +1 more
TL;DR: The aim of this Minireview is to provide an evaluation of the state of knowledge on the functional consequences of CAD-induced phospholipidosis.
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In vitro chemopreventive effects of plant polysaccharides (Aloe barbadensis miller, Lentinus edodes, Ganoderma lucidum and Coriolus versicolor).
TL;DR: Results suggest that some plant polysaccharides produced both anti-genotoxic and anti-tumor promoting activities in in vitro models and, therefore, might be considered as potential agents for cancer chemoprevention.
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Role of rat strain in the differential sensitivity to pharmaceutical agents and naturally occurring substances.
Sam Kacew,Michael F. W. Festing +1 more
TL;DR: Genetically determined differences in the responsiveness of rat strains to drugs and naturally occurring chemicals are summarized to show that susceptibility is dependent on the target organ sensitivities, which may also be strain dependent.
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An evaluation of possible mechanisms underlying amiodarone-induced pulmonary toxicity.
Mark J. Reasor,Sam Kacew +1 more
TL;DR: With a clearer understanding of the possible contributions of these mechanisms to AIPT, it may be possible to develop strategies to alleviate toxicity and prolong the usefulness of amiodarone in the treatment of cardiac arrhythmias.