S
Sandrine Prost
Researcher at University of Edinburgh
Publications - 24
Citations - 967
Sandrine Prost is an academic researcher from University of Edinburgh. The author has contributed to research in topics: DNA damage & Nucleotide excision repair. The author has an hindex of 16, co-authored 24 publications receiving 723 citations. Previous affiliations of Sandrine Prost include Queen's University & Edinburgh Cancer Research Centre.
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Journal ArticleDOI
Tissue-specific Immunopathology in Fatal COVID-19.
David A. Dorward,Clark D Russell,In Hwa Um,Mustafa Elshani,Stuart D. Armstrong,Rebekah Penrice-Randal,Tracey Millar,Chris E B Lerpiniere,Giulia Tagliavini,Catherine Hartley,Nadine Randle,Naomi N. Gachanja,Philippe M D Potey,Xiaofeng Dong,Alison M Anderson,Victoria L Campbell,Alasdair J Duguid,Wael Al Qsous,Ralph BouHaidar,J Kenneth Baillie,Kevin Dhaliwal,William A Wallace,Christopher Bellamy,Sandrine Prost,Colin Smith,Julian A. Hiscox,Julian A. Hiscox,Julian A. Hiscox,David J. Harrison,Christopher D. Lucas +29 more
TL;DR: Tissue-specific immunopathology occurs in COVID-19, implicating a significant component of the immune-mediated, virus-independent immunopathologic process as a primary mechanism in severe disease.
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Functional Immune Anatomy of the Liver-As an Allograft.
Anthony J. Demetris,Christopher Bellamy,Chandrashekhar R. Gandhi,Sandrine Prost,Yasuni Nakanuma,Donna B. Stolz +5 more
TL;DR: A better understanding is provided of liver immune microanatomy and physiology and thereby of the potential structural consequences of low‐level, including allo‐antibody‐mediated injury; and how liver allografts modulate immune reactions.
Journal ArticleDOI
Hepatitis B x Protein Inhibits p53-dependent DNA Repair in Primary Mouse Hepatocytes
TL;DR: In this article, the levels of global repair (removal of cyclobutane pyrimidine dimers and 6-4 photoproducts) and transcription-coupled repair were studied in primary wild-type and p53-null mouse hepatocytes.
Journal ArticleDOI
Monocyte-derived macrophages promote breast cancer bone metastasis outgrowth.
Ruoyu Ma,Hui Zhang,Xue-Feng Li,Cheng-Bin Zhang,Cigdem Selli,Giulia Tagliavini,Alyson D. Lam,Sandrine Prost,Andrew H. Sims,Hai Yang Hu,Tianlei Ying,Zhan Wang,Zhaoming Ye,Jeffrey W. Pollard,Jeffrey W. Pollard,Bin-Zhi Qian,Bin-Zhi Qian +16 more
TL;DR: This study identifies a novel population of CD204+IL4R+ bone metastasis–associated macrophages (BoMAMs) in mouse models and patient samples that significantly promote metastatic outgrowth of breast cancer in vivo.
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Microarray analysis of gene expression of mouse hepatocytes of different ploidy
TL;DR: The results show that polyploid hepatocytes are stable and “normal” without aberrant gene expression, unlike what is thought for cancer cells, and support the hypothesis that hepatocytepolyploidisation is a protective mechanism against oxidative stress that occurs via a controlled process throughout growth and aging where binucleation is important.