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Sang Won Suh

Researcher at Hallym University

Publications -  87
Citations -  7784

Sang Won Suh is an academic researcher from Hallym University. The author has contributed to research in topics: Neuron death & Hippocampal formation. The author has an hindex of 28, co-authored 75 publications receiving 6931 citations. Previous affiliations of Sang Won Suh include University of California, San Francisco.

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Protective Effects of Protocatechuic Acid on Seizure-Induced Neuronal Death

TL;DR: It is found that, compared to the seizure vehicle-treated group, PCA administration reduced neuronal death and oxidative stress in the hippocampus and seizure-induced microglia activation was significantly reduced by the PCA treatment.
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Impairment of autophagic flux promotes glucose reperfusion-induced neuro2A cell death after glucose deprivation.

TL;DR: The results suggest that interplay of autophagy, caspase3 activation and lysosomal proteases serve as a basis for neuronal death after hypoglycemia, and provide the molecular mechanism of neuronal death by glucose reperfusion.
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Decreased cysteine uptake by EAAC1 gene deletion exacerbates neuronal oxidative stress and neuronal death after traumatic brain injury

TL;DR: It is indicated that cysteine uptake by EAAC1 is important for neuronal antioxidant function and survival following TBI, and suggests that administration of NAC has therapeutic potential in preventing TBI-induced neuronal death.
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Carvacrol Attenuates Hippocampal Neuronal Death after Global Cerebral Ischemia via Inhibition of Transient Receptor Potential Melastatin 7

TL;DR: The present study found that administration of carvacrol significantly decreased the number of degenerating neurons, microglial activation, oxidative damage, and zinc translocation after GCI, via downregulation of TRPM7 channels.
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Effect of Adipose-Derived Mesenchymal Stem Cell Administration and Mild Hypothermia Induction on Delayed Neuronal Death After Transient Global Cerebral Ischemia

TL;DR: Administration of mesenchymal stem cells after transient global cerebral ischemia has a prominent protective effect on delayed neuron death, even compared with mild hypothermia.