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Soojin Hwang

Researcher at Ewha Womans University

Publications -  8
Citations -  234

Soojin Hwang is an academic researcher from Ewha Womans University. The author has contributed to research in topics: Phosphorylation & Nitric oxide. The author has an hindex of 8, co-authored 8 publications receiving 202 citations.

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Journal ArticleDOI

Uric acid attenuates nitric oxide production by decreasing the interaction between endothelial nitric oxide synthase and calmodulin in human umbilical vein endothelial cells: A mechanism for uric acid-induced cardiovascular disease development

Jung Hyun Park, +5 more
- 01 Aug 2013 - 
TL;DR: It is suggested that uric acid decreased eNOS activity and NO production through reducing the binding between eN OS and CaM in EC and this result may provide molecular mechanism by which uric Acid induces endothelial dysfunction.
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CCN1 Secreted by Tonsil‐Derived Mesenchymal Stem Cells Promotes Endothelial Cell Angiogenesis via Integrin αvβ3 and AMPK

Yoon Shin Park, +8 more
- 01 Jan 2015 - 
TL;DR: It is demonstrated that T‐MSC increase the secretion of CCN1 in response to endodermal differentiation and that integrin αvβ3 and AMPK mediateCCN1‐induced EC migration and tube formation independent of intracellular ATP levels alteration.
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Far-infrared radiation inhibits proliferation, migration, and angiogenesis of human umbilical vein endothelial cells by suppressing secretory clusterin levels.

Soojin Hwang, +4 more
- 28 Apr 2014 - 
TL;DR: A novel mechanism by which FIR radiation inhibits the proliferation, migration, and angiogenesis of HUVEC, via decreasing sCLU is demonstrated.
Journal ArticleDOI

Histone H3 lysine 27 and 9 hypermethylation within the Bad promoter region mediates 5-Aza-2′-deoxycytidine-induced Leydig cell apoptosis: implications of 5-Aza-2′-deoxycytidine toxicity to male reproduction

Ji-Young Choi, +7 more
- 01 Jan 2013 - 
TL;DR: It is reported that 5-Aza increased the apoptotic protein Bad epigenetically in the testosterone-producing mouse TM3 Leydig cell line, demonstrating for the first time that Bad expression is regulated at least by EZH2-mediated H3K27-3me or G9a-like protein/euchromatic histone methyltransferase 1 (GLP/Eu-HMTase1)-mediated H 3K9- 3me in mouse TM 3 Le
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Trichostatin A epigenetically increases calpastatin expression and inhibits calpain activity and calcium‐induced SH‐SY5Y neuronal cell toxicity

Jungwon Seo, +9 more
- 01 Dec 2013 - 
TL;DR: In this paper, trichostatin A (TSA) was shown to attenuate calcium-induced neuronal cell death by the inhibition of calpain activity which is mediated in part by increased calpastatin expression via histone hyperacetylation within the promoter region.