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Stefania Bilotto

Researcher at National Research Council

Publications -  21
Citations -  1275

Stefania Bilotto is an academic researcher from National Research Council. The author has contributed to research in topics: Cancer & Quercetin. The author has an hindex of 12, co-authored 21 publications receiving 1090 citations. Previous affiliations of Stefania Bilotto include University of Basilicata & Centre national de la recherche scientifique.

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Journal ArticleDOI

The flavonoid quercetin in disease prevention and therapy: facts and fancies.

TL;DR: Quercetin can be considered the prototype of a naturally-occurring chemopreventive agent because of its key roles in triggering the "hallmarks of cancer" and several critical points must be taken into account when considering the potential therapeutic use of this molecule.
Book ChapterDOI

Quercetin: A Pleiotropic Kinase Inhibitor Against Cancer

TL;DR: The ability of the molecule to inhibit protein kinases involved in deregulated cell growth in cancer cells is reviewed, and the use of natural or synthetic compounds able to stop, reverse, or delay the process of tumorigenesis in its early stages is reviewed.
Journal ArticleDOI

Dietary polyphenols in cancer prevention: the example of the flavonoid quercetin in leukemia

TL;DR: Considering the low toxicity of the flavonoids toward normal peripheral blood cells, the experimental results are in favor of a potential use of quercetin in adjuvant chemotherapy in CLL or other types of cancer.
Journal ArticleDOI

Omega-3 polyunsaturated fatty acids and cancer: lessons learned from clinical trials

TL;DR: This review has collected the available clinical data on the therapeutic role of omega-3 FAs against breast cancer, colorectal cancer, leukemia, gastric cancer, pancreatic cancer, esophagealcancer, prostate cancer, lung cancer, head and neck cancer, as well as cancer cachexia.
Journal ArticleDOI

Quercetin reduced inflammation and increased antioxidant defense in rat adjuvant arthritis.

TL;DR: Results indicated that quercetin was able to ameliorate all markers of inflammation and oxidative stress measured and inhibited the 2-fold increase of NF-қB activity observed in lung, liver and joint after induction of arthritis.