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Stefania D'Angelo

Researcher at University of Naples Federico II

Publications -  68
Citations -  1946

Stefania D'Angelo is an academic researcher from University of Naples Federico II. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 20, co-authored 41 publications receiving 1546 citations. Previous affiliations of Stefania D'Angelo include Parthenope University of Naples & Seconda Università degli Studi di Napoli.

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BPA and Nutraceuticals, Simultaneous Effects on Endocrine Functions.

TL;DR: Dietary remediation may represent a successful therapeutic approach to maintain and preserve health against BPA damage and the use of nutraceuticals can be considered a support for clinical treatment.
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Current Evidence on the Effect of Dietary Polyphenols Intake on Brain Health

TL;DR: Key questions to improve the coherence and reproducibility in the development ofpolyphenols as a possible future therapeutic drug require a better understanding of the sources of polyphenols, their treatment and more standardized tests including bioavailability of bioactive metabolites and studies of permeability of the brain.
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Segmented poly(ether–ester–amide)s based on poly(l,l-lactide) macromers

TL;DR: In this article, Sebacoyl chloride (SEB), α,ω-hydroxyl terminated poly( l, l -lactide) (PLLA) or PLLA-PEG-PLLa macromers and the hydrophilic diamines 4,7,10-trioxa-1,13-tridecanediamine (1 ) and 3,6,9,12,15-pentoxa -1,17-heptadecanedienine (2 ) or diamide-d
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Cytoskeletal behaviour in spectrin and in band 3 deficient spherocytic red cells: evidence for a differentiated splenic conditioning role

TL;DR: The results support the notion that band 3 deficient erythrocytes are not affected by an extensive cytoskeletal derangement and find a remarkable increase of membrane methylation in the unsplenectomized, spectrin‐deficient, HS patients, suggesting a striking membrane skeleton disarray.
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Accumulation of altered aspartyl residues in erythrocyte membrane proteins from patients with sporadic amyotrophic lateral sclerosis.

TL;DR: The data show that, in vitro assay, abnormal IsoAsp residues were significantly higher in ALS patients erythrocyte membrane proteins with an increased methyl accepting capability relative to controls, which is probably related to a reduced efficiency of the S-adenosylmethionine (AdoMet)-dependent repair system causing increased protein instability at Asn sites.