S
Sten Wingren
Researcher at Linköping University
Publications - 55
Citations - 2482
Sten Wingren is an academic researcher from Linköping University. The author has contributed to research in topics: Breast cancer & Cancer. The author has an hindex of 24, co-authored 55 publications receiving 2441 citations. Previous affiliations of Sten Wingren include Health Science University & Örebro University.
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Genetic variants of CYP3A5, CYP2D6, SULT1A1, UGT2B15 and tamoxifen response in postmenopausal patients with breast cancer
TL;DR: The present data suggest that genetic variation in CYP3A5 may predict response to tamoxifen therapy, which is a combination of several mechanisms behind the resistance of oestrogen-receptor-positive patients with breast cancer.
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Genotype of metabolic enzymes and the benefit of tamoxifen in postmenopausal breast cancer patients.
TL;DR: The present study suggests that genotype of metabolic enzymes might be useful as a guide for adjuvant endocrine treatment of postmenopausal breast cancer patients, however, results are in contradiction to prior hypotheses and the present sample size is relatively small.
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Prognostic significance of cytoplasmic p53 oncoprotein in colorectal adenocarcinoma.
Xiao-Feng Sun,John Carstensen,Olle Stål,Sten Wingren,Thomas Hatschek,Bo Nordenskjöld,Hongmei Zhang +6 more
TL;DR: It is concluded that cytoplasmic expression of p53 may be a useful biological indicator of prognosis in colorectal adenocarcinoma.
Journal Article
Association of Breast Cancer Progression with a Vitamin D Receptor Gene Polymorphism
TL;DR: It is concluded that polymorphism in the VDR gene may influence tumor progression and tamoxifen treatment response in early-onset breast carcinomas.
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c-erbB-2 expression and benefit from adjuvant chemotherapy and radiotherapy of breast cancer.
O Stål,Siw Sullivan,Sten Wingren,Lambert Skoog,Lars Erik Rutqvist,John Carstensen,Bo Nordenskjöld +6 more
TL;DR: Frozen tissue from primary tumours of 152 premenopausal breast cancer patients, who participated in a trial comparing radiotherapy with adjuvant chemotherapy (cyclophosphamide, methotrexate, 5-fluorouracil, CMF), was analysed for c-erbB-2 protein expression, measured by flow cytometry.