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Stephanie Reed

Researcher at University of California, San Diego

Publications -  38
Citations -  1318

Stephanie Reed is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Entamoeba histolytica & Leishmania. The author has an hindex of 18, co-authored 38 publications receiving 1292 citations. Previous affiliations of Stephanie Reed include Washington University in St. Louis & University of Cambridge.

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Aberrant protamine 1/protamine 2 ratios in sperm of infertile human males

TL;DR: The distribution of protamines in sperm obtained from a select group of infertile males producing an elevated level of large sperm heads, in contrast, was different from that of the fertile males.
Journal Article

Cell mediated immunity in American cutaneous and mucosal leishmaniasis.

TL;DR: The studies have determined that the parameters of lymphocyte and macrophage functions evaluated in ML and CL patients are comparable, except for an enhanced lymphoproliferative response, with leishmania antigen in ML patients.
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Mechanism of resistance to complement-mediated killing of bacteria encoded by the Salmonella typhimurium virulence plasmid gene rck.

TL;DR: It is found that pADEO16, a recombinant cosmid carrying the rck gene of the Salmonella typhimurium virulence plasmid, when cloned into either rough or smooth Escherichia coli andSalmonella strains, confers high level resistance to the bactericidal activity of pooled normal human serum.
Journal Article

Transforming growth factor-beta in human cutaneous leishmaniasis.

TL;DR: Observations suggest an important role for TGF-beta in human leishmaniasis, with its production by infected macrophages being probably related to parasite establishment in the early stages of the disease.
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Cloning of a virulence factor of Entamoeba histolytica. Pathogenic strains possess a unique cysteine proteinase gene.

TL;DR: Analysis of the predicted amino acid sequence of the ACP1 proteinase gene reveals homology with cysteine proteinases released by activated macrophages and invasive cancer cells, suggesting an evolutionarily conserved mechanism of tissue invasion.