S
Sudipta Chakraborty
Researcher at Vanderbilt University Medical Center
Publications - 13
Citations - 726
Sudipta Chakraborty is an academic researcher from Vanderbilt University Medical Center. The author has contributed to research in topics: Neurodegeneration & Caenorhabditis elegans. The author has an hindex of 11, co-authored 13 publications receiving 591 citations. Previous affiliations of Sudipta Chakraborty include Vanderbilt University.
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Journal ArticleDOI
Manganese homeostasis in the nervous system.
Pan Chen,Sudipta Chakraborty,Somshuvra Mukhopadhyay,Eun Sook Lee,Monica Maria Bastos Paoliello,Aaron B. Bowman,Michael Aschner,Michael Aschner +7 more
TL;DR: This review will focus on current studies related to mechanisms underlying Mn import and export, primarily the Mn transporters, and their function and roles in Mn‐induced neurotoxicity.
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Cellular transport and homeostasis of essential and nonessential metals
TL;DR: This review provides an overview of essential and nonessential metal transport and homeostatic processes.
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The effects of pdr1, djr1.1 and pink1 loss in manganese-induced toxicity and the role of α-synuclein in C. elegans
Julia Bornhorst,Julia Bornhorst,Sudipta Chakraborty,Sören Meyer,Hanna Lohren,Sigrid Grosse Brinkhaus,Adam L. Knight,Kim A. Caldwell,Guy A. Caldwell,Uwe Karst,Tanja Schwerdtle,Aaron B. Bowman,Michael Aschner,Michael Aschner +13 more
TL;DR: A novel, neuroprotective role for WT human α-synuclein is uncovered in attenuating Mn-induced toxicity in the background of PD-associated genes, and the role of extracellular dopamine in exacerbating Mn neurotoxicity is supported.
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Metal-induced neurodegeneration in C. elegans
TL;DR: This review discusses methods to assess degeneration as well as outlines techniques for genetic manipulation and presents a comprehensive survey of the existing literature on metal-induced degeneration studies in the worm.
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Oxidative stress mechanisms underlying Parkinson's disease-associated neurodegeneration in C. elegans.
TL;DR: The roles of C. elegans homologs for the oxidative stress-associated transcription factor Nrf2, as well as the autosomal recessive, early-onset Parkinson’s disease (PD)-associated proteins Parkin, DJ-1, and PINK1, in neurodegenerative processes are focused on.