Tamara V. Gurvits

TL;DR: Hippocampal volume was directly correlated with combat exposure, which suggests that traumatic stress may damage the hippocampus and smaller hippocampi volume may be a pre-existing risk factor for combat exposure and/or the development of PTSD upon combat exposure.

TL;DR: It is suggested that memory functioning represents a neurocognitive domain of specific relevance to the development of PTSD in trauma-exposed individuals, which can be distinguished from generalized attentional impairment as well as the effects of trauma exposure severity, IQ, comorbid depression, history of alcohol use, and history of developmental learning problems.

TL;DR: Cognitive performance in monozygotic twin pairs who were discordant for combat exposure was evaluated to support the notion that specific domains of cognitive function may serve as premorbid risk or protective factors in PTSD.

TL;DR: Analysis of pairs of Vietnam combat veterans and their noncombat‐exposed, identical twins supports the conclusion that the latter abnormalities represent antecedent, familial vulnerability factors for developing chronic PTSD upon exposure to a traumatic event.

TL;DR: Increased aggression in war veterans is more appropriately regarded as a property of PTSD, rather than a direct consequence of military combat, and the association between compromised neurologic and neuropsychologic status and the psychometric measures was modest and explained little of the group differences.