scispace - formally typeset
T

Tanja Maria Michel

Researcher at University of Southern Denmark

Publications -  60
Citations -  2012

Tanja Maria Michel is an academic researcher from University of Southern Denmark. The author has contributed to research in topics: Autism & Autism spectrum disorder. The author has an hindex of 23, co-authored 53 publications receiving 1689 citations. Previous affiliations of Tanja Maria Michel include Odense University Hospital & Tabriz University of Medical Sciences.

Papers
More filters
Journal ArticleDOI

Brain structure anomalies in autism spectrum disorder--a meta-analysis of VBM studies using anatomic likelihood estimation.

TL;DR: This unbiased summary provided evidence for consistent structural abnormalities in spite of heterogeneous diagnostic criteria and voxel‐based morphometry (VBM) methodology, but also hinted at a dependency of VBM findings on the age of the patients.
Journal ArticleDOI

The Role of Oxidative Stress in Depressive Disorders

TL;DR: A comprehensive overview over the current literature discussing the involvement of oxidative stress and free radicals in depression is given, finding numerous reports elaborating depressive disorder and oxidative stress.
Journal ArticleDOI

Evidence for oxidative stress in the frontal cortex in patients with recurrent depressive disorder--a postmortem study.

TL;DR: Compared manganese (Mn) and copper/zinc (Cu/Zn) superoxide dismutase (SOD) coenzyme concentrations in postmortem PFC and hippocampal brain tissue from 7 patients with recurrent depressive disorder and 7 neuropsychiatrically healthy controls using sandwich-type enzyme-linked immunosorbent assay (ELISA) technique.
Journal ArticleDOI

Regulatory T cells increased while IL-1β decreased during antidepressant therapy

TL;DR: The increase in CD4(+)CD25(hi) Tregs during antidepressant therapy may be the reason for the decrease in cytokine production and the recovery from depression.
Journal ArticleDOI

The role of neurotrophic factors in autism

TL;DR: The role of NTFs in ASD is explored and evidence derived from neurochemical investigations, gene association studies and animal models are included to further elucidate the puzzling aetiology of these conditions.