T
Terrence Deak
Researcher at Binghamton University
Publications - 143
Citations - 7218
Terrence Deak is an academic researcher from Binghamton University. The author has contributed to research in topics: Corticosterone & Hypothalamus. The author has an hindex of 43, co-authored 134 publications receiving 6484 citations. Previous affiliations of Terrence Deak include University of Colorado Boulder & State University of New York System.
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Journal ArticleDOI
Sex differences in dendritic atrophy of ca3 pyramidal neurons in response to chronic restraint stress
Liisa A.M. Galea,Bruce S. McEwen,Patima Tanapat,Terrence Deak,Robert L. Spencer,Firdaus S. Dhabhar +5 more
TL;DR: It appears unlikely that sex differences in the corticosteroid-binding globulin and corticosterone response can account for these morphological differences found in males and females.
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Exposure to acute stress induces brain interleukin-1beta protein in the rat
Kien T. Nguyen,Terrence Deak,Stephanie M. Owens,Tadahiko Kohno,Monika Fleshner,Linda R. Watkins,Steven F. Maier +6 more
TL;DR: Elimination of the stress-induced rise in corticosterone unmasked a robust and widespread increase in brain IL-1β, possibly suppressed by the rapid and prolonged high levels of glucocorticoids produced by IS.
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Treadmill running produces both positive and negative physiological adaptations in Sprague-Dawley rats.
TL;DR: T treadmill running produces both positive training adaptations and potentially negative adaptations that are indicative of chronic stress and that these changes could potentially impact other measures of interest.
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A users guide to HPA axis research.
Robert L. Spencer,Terrence Deak +1 more
TL;DR: This Users Guide is intended to provide a fundamental overview of conceptual, technical and practical knowledge that will assist individuals who engage in and evaluate HPA axis research.
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Prior Stressor Exposure Sensitizes LPS-Induced Cytokine Production
TL;DR: It is suggested that exposure to inescapable tailshock sensitizes the innate immune response to LPS by resulting in either a larger or a more rapid induction of proinflammatory cytokines.