Kevin O'Connor

TL;DR: The present studies demonstrate that both ipsilateral and mirror-image SIN-induced allodynias are reversed by intrathecal (peri-spinal) delivery of fluorocitrate, a glial metabolic inhibitor, and provide the first evidence that ipsilaterally and Mirror-image inflammatory neuropathy pain are created both acutely and chronically through glial and proinflammatory cytokine actions.

TL;DR: This article developed an analytical framework called "Becoming an Engineer" that focuses upon changes occurring over time as students traverse their undergraduate educations in engineering, including disciplinary knowledge, identification, and navigation.

TL;DR: The results support the concept that activated glia in dorsal spinal cord can create exaggerated pain states via the release of proinflammatory cytokines through activation of microglia and astrocytes.

TL;DR: These initial investigations of spinal fractalkine effects suggest that exogenous and endogenous fractalkines are involved in spinal sensitization, including that induced by peripheral neuropathy.

TL;DR: It is suggested that exposure to inescapable tailshock sensitizes the innate immune response to LPS by resulting in either a larger or a more rapid induction of proinflammatory cytokines.