Thomas Pfannschmidt

TL;DR: Here it is shown that the redox state of plastoquinone also controls the rate of transcription of genes encoding reaction-centre apoproteins of photosystem I and photosystem II, and the stoichiometry between the two photosystems changes in a way that counteracts the inefficiency produced when either photosystem limits the rates of the other.

TL;DR: The results are consistent with transcriptional effects observed previously with chloroplasts isolated in vitro and demonstrate that redox control of chloroplast transcription initiates long‐term adjustments that compensate for imbalance in energy distribution and adapt the whole plant to altered light environments.

TL;DR: Both activation/inactivation and differential gene expression seem to play a role in the regulation of the plastid transcription machinery.

TL;DR: Using a homologous in vitro system, it is found that PTK selectively phosphorylates subunit(s) of plastid RNA polymerase and is involved in determining the level of faithful transcription from the chloroplast psbA promoter, establishing a role for phosphorylation and redox state in the regulation ofplastid gene expression.

TL;DR: Evidence is provided that the A enzyme can be converted into a Rif-sensitive enzyme form in a phosphorylation-dependent manner in vitro by a treatment that results in depletion of a beta-like subunit.