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Timothy M. Greenaway

Researcher at University of Tasmania

Publications -  56
Citations -  2335

Timothy M. Greenaway is an academic researcher from University of Tasmania. The author has contributed to research in topics: Diabetes mellitus & Multiple endocrine neoplasia. The author has an hindex of 24, co-authored 53 publications receiving 2145 citations. Previous affiliations of Timothy M. Greenaway include Hobart Corporation & Deakin University.

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Brain atrophy in type 2 diabetes: regional distribution and influence on cognition.

TL;DR: T2DM was associated with poorer visuospatial construction, planning, visual memory, and speed independent of age, sex, education, and vascular risk factors and the strength of these associations was attenuated when adjusted for hippocampal and total gray volumes but was unchanged by adjustment for cerebrovascular lesions or white matter volume.
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Expression of the MEN-1 gene in a large kindred with multiple endocrine neoplasia type 1

TL;DR: expression of the MEN‐1 gene in a large kindred with multiple endocrine neoplasia type 1 (Minisymposium: MEN & VHL) is described.
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Adrenal lesions in a large kindred with multiple endocrine neoplasia type 1.

TL;DR: Primary hypersecretory syndromes of the adrenal glands appear to be rare, and the majority of lesions follow an indolent clinical course.
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Vitamin D insufficiency in adolescent males in Southern Tasmania: prevalence, determinants, and relationship to bone turnover markers.

TL;DR: Vitamin D insufficiency is common in healthy adolescent boys in winter in this setting, is primarily derived from sports-related sun exposure, and is associated with bone turnover markers, suggesting that a 25(OH)D3 level of at least 43–55 nmol/l is required for optimal bone health in children.
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Osteoporosis in Multiple Endocrine Neoplasia Type 1: Severity, Clinical Significance, Relationship to Primary Hyperparathyroidism, and Response to Parathyroidectomy

TL;DR: Despite difficulty in achieving a cure of PHPT in MEN 1, parathyroidectomy has an important role in the optimization of BMD for patients with MEN 1.