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Velandai Srikanth

Researcher at Monash University

Publications -  312
Citations -  12746

Velandai Srikanth is an academic researcher from Monash University. The author has contributed to research in topics: Population & Stroke. The author has an hindex of 53, co-authored 266 publications receiving 9982 citations. Previous affiliations of Velandai Srikanth include Florey Institute of Neuroscience and Mental Health & Menzies Research Institute.

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A meta-analysis of sex differences prevalence, incidence and severity of osteoarthritis

TL;DR: The results demonstrate the presence of sex differences in OA prevalence and incidence, with females generally at a higher risk and females also tend to have more severe knee OA, particularly after menopausal age.
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Advanced glycation endproducts and their receptor RAGE in Alzheimer's disease.

TL;DR: In this paper, the authors revisited the hypothesis that advanced glycation endproducts (AGEs) and their receptor RAGE may play an important role in disease pathogenesis by influencing transport of β-amyloid into the brain or by manipulating inflammatory mechanisms.
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Quality of Life After Stroke: The North East Melbourne Stroke Incidence Study (NEMESIS)

TL;DR: Interventions targeting handicap and mood have the potential to improve HRQoL independently of physical impairment and disability.
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Brain atrophy in type 2 diabetes: regional distribution and influence on cognition.

TL;DR: T2DM was associated with poorer visuospatial construction, planning, visual memory, and speed independent of age, sex, education, and vascular risk factors and the strength of these associations was attenuated when adjusted for hippocampal and total gray volumes but was unchanged by adjustment for cerebrovascular lesions or white matter volume.
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Genetic contributions to variation in general cognitive function: a meta-analysis of genome-wide association studies in the CHARGE consortium (N=53 949)

Gary Davies, +151 more
- 01 Feb 2015 - 
TL;DR: In hypothesis-driven tests, there was significant association between general cognitive function and four genes previously associated with Alzheimer’s disease: TOMM40, APOE, ABCG1 and MEF2C.