T
Tyrone D. Cannon
Researcher at Yale University
Publications - 571
Citations - 44590
Tyrone D. Cannon is an academic researcher from Yale University. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 105, co-authored 535 publications receiving 39587 citations. Previous affiliations of Tyrone D. Cannon include University of Pennsylvania & University of California, Los Angeles.
Papers
More filters
Journal ArticleDOI
Early traumatic experiences in those at clinical high risk for psychosis.
Jean Addington,Jacqueline Stowkowy,Kristin S. Cadenhead,Barbara A. Cornblatt,Thomas H. McGlashan,Diana O. Perkins,Larry J. Seidman,Ming T. Tsuang,Elaine F. Walker,Scott W. Woods,Tyrone D. Cannon,Tyrone D. Cannon +11 more
TL;DR: The purpose of this study was to determine the extent of childhood trauma and bullying in young people at clinical high risk (CHR) of developing psychosis.
Journal ArticleDOI
Phenomenology and Functioning in First-Episode Schizophrenia
Derri L. Shtasel,Raquel E. Gur,Fiona Gallacher,Carolyn Heimberg,Tyrone D. Cannon,Ruben C. Gur +5 more
TL;DR: The symptom profile of schizophrenia exists at the outset, that negative symptoms are associated with poor premorbid and current functioning, but that the role of positive symptoms is more complex and may vary in subtypes.
Journal ArticleDOI
A Twin Study of Genetic Contributions to Hippocampal Morphology in Schizophrenia
Katherine L. Narr,Theo G.M. van Erp,Tyrone D. Cannon,Roger P. Woods,Paul M. Thompson,Seonah Jang,Rebecca E. Blanton,Veli-Pekka Poutanen,Matti O. Huttunen,Jouko Lönnqvist,Carl-Gustav Standerksjöld-Nordenstam,Jaakko Kaprio,John C. Mazziotta,Arthur W. Toga +13 more
TL;DR: Results suggest that hippocampal volume reduction may be a trait marker for identifying individuals possessing a genetic predisposition for schizophrenia.
Journal ArticleDOI
Hippocampal activations during encoding and retrieval in a verbal working memory paradigm.
TL;DR: An analysis of retrieval activation separated by probe type showed a trend toward greater bilateral hippocampal activation for probes related to the target than for unrelated probes and still greater activation for target matches, suggesting that there may be roles for the hippocampus and DLPFC in working memory that change as function of information processing stage.