V
V. Sotosek Tokmadzic
Researcher at University of Rijeka
Publications - 5
Citations - 153
V. Sotosek Tokmadzic is an academic researcher from University of Rijeka. The author has contributed to research in topics: Cytotoxic T cell & Inflammation. The author has an hindex of 4, co-authored 5 publications receiving 143 citations.
Papers
More filters
Journal ArticleDOI
The presence of functional mannose receptor on macrophages at the maternal–fetal interface
Gordana Laškarin,Kristijan Ćupurdija,V. Sotosek Tokmadzic,Dorotea Dorčić,J. Dupor,Koraljka Juretić,Natasa Strbo,T. Bogovic Crncic,F. Marchezi,Paola Allavena,Alberto Mantovani,Lj Randic,Daniel Rukavina +12 more
TL;DR: Data indicate that the MR+ macrophages, surrounding early decidual glands, are able to internalize ligands for carbohydrate recognition domain of the receptor, including decidUAL secretory phase mucin TAG-72.
Journal ArticleDOI
Early changes in frequency of peripheral blood lymphocyte subpopulations in severe traumatic brain-injured patients.
Ines Mrakovčić-Šutić,V. Sotosek Tokmadzic,Gordana Laškarin,Hana Mahmutefendić,Pero Lučin,Zeljko Zupan,Alan Šustić +6 more
TL;DR: Investigation of changes in frequency of leucocytes subpopulations in peripheral blood of patients with STBI during the course of intensive care treatment found a decrease in cells with cytotoxic phenotype, which might explain high incidence of susceptibility to infection of patientswith STBI.
Journal ArticleDOI
Perforin expression in peripheral blood lymphocytes and skin-infiltrating cells in patients with lichen planus.
L. Prpic Massari,Marija Kaštelan,F. Gruber,Gordana Laškarin,V. Sotosek Tokmadzic,Natasa Strbo,Gordana Zamolo,Gordana Zauhar,Daniel Rukavina +8 more
TL;DR: This data indicates that the placenta of lichen planus is a T‐cell‐mediated autoimmune disease in which cytotoxic mechanisms have been poorly investigated and the mechanisms responsible for cell reprograming are unknown.
Journal ArticleDOI
Harmful immune reactions during acute myocardial infarction
TL;DR: In vivo tests are proposed to be used to measure endothelial dysfunction, as well as ventricular dysfunction by ultrasound methods, and their correlation with immunological and/or biochemical parameters, to hypothesize that this unwanted side effect of increase in proinflammatory mediators can lead to harmful systemic immune reactions directed towards various dysfunctional endothelia.
Journal ArticleDOI
Corrigendum to "Endothelial dysfunction mediated by interleukin-18 in patients with ischemic heart disease undergoing coronary artery bypass grafting surgery" [Med. Hypotheses 104 (2017) 20-24].
Jadranko Sokolić,V. Sotosek Tokmadzic,Danijel Knezevic,Igor Medved,N. Vukelic Damjani,Sanja Balen,Marijana Rakić,A. Lanca Bastiancic,Gordana Laškarin +8 more