V
Valentina Gentili
Researcher at University of Ferrara
Publications - 59
Citations - 975
Valentina Gentili is an academic researcher from University of Ferrara. The author has contributed to research in topics: Medicine & Human leukocyte antigen. The author has an hindex of 14, co-authored 49 publications receiving 564 citations.
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Journal ArticleDOI
TLR3 and TLR7 RNA Sensor Activation during SARS-CoV-2 Infection.
Daria Bortolotti,Valentina Gentili,Sabrina Rizzo,Giovanna Schiuma,Silvia Beltrami,Giovanni Strazzabosco,M. Fernandez,Francesca Caccuri,Arnaldo Caruso,Roberta Rizzo +9 more
TL;DR: In this article, the authors investigated the role of pathogen-associated molecular patterns (PAMPs)-pattern recognition receptors (PRRs) interaction and showed that both TLR3 and TLR7 are involved in the control of innate immunity during lung SARS-CoV-2 infection.
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Presence of HHV-6A in Endometrial Epithelial Cells from Women with Primary Unexplained Infertility
Roberto Marci,Valentina Gentili,Daria Bortolotti,Giuseppe Lo Monte,Elisabetta Caselli,Silvia Bolzani,Antonella Rotola,Dario Di Luca,Roberta Rizzo +8 more
TL;DR: It is indicated, for the first time, that HHV-6A infection might be an important factor in female unexplained infertility development, with a possible role in modifying endometrial NK cells immune profile and ability to sustain a successful pregnancy.
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SARS-CoV-2 Spike 1 Protein Controls Natural Killer Cell Activation via the HLA-E/NKG2A Pathway.
TL;DR: It is shown for the first time that NK cells are affected by SP1 expression in lung epithelial cells via HLA-E/NKG2A interaction and the resulting NK cells’ exhaustion might contribute to immunopathogenesis in SARS-CoV-2 infection.
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Human herpesvirus 7, Epstein-Barr virus and human cytomegalovirus in periodontal tissues of periodontally diseased and healthy subjects.
Antonella Rotola,Enrico Cassai,Roberto Farina,Elisabetta Caselli,Valentina Gentili,Tiziana Lazzarotto,Leonardo Trombelli +6 more
TL;DR: The results indicate that gingival tissues can be considered a potential reservoir for HHV-7 and the presence of EBV seems to be associated with the diseased state of the patient and site.
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HHV-6A infection induces amyloid-beta expression and activation of microglial cells
TL;DR: Microglial cells are permissive to HHV-6A infection that induces the expression of Aβ and an activation status, determined by TREM2, ApoE, cytokines, and tau expression.