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Wei Li

Researcher at Southwest University

Publications -  6
Citations -  206

Wei Li is an academic researcher from Southwest University. The author has contributed to research in topics: Gene & Sclerotinia sclerotiorum. The author has an hindex of 4, co-authored 4 publications receiving 132 citations.

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Genome-wide association analysis and differential expression analysis of resistance to Sclerotinia stem rot in Brassica napus.

TL;DR: Genes found to be specific to the R genotype related to the jasmonic acid pathway, lignin biosynthesis, defence response, signal transduction and encoding transcription factors, including a tau class glutathione S-transferase (GSTU) gene cluster are identified.
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Genetic and transcriptomic analyses of lignin- and lodging-related traits in Brassica napus

TL;DR: In this study, it is found that the syringyl/guaiacyl (S/G) monolignol ratio exhibits a significant negative correlation with disease and lodging resistance, indicating crosstalk exists between glucosinolate metabolic processes and lignin biosynthesis.
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Cloning and Phylogenetic Analysis of Brassica napus L. Caffeic Acid O-Methyltransferase 1 Gene Family and Its Expression Pattern under Drought Stress.

TL;DR: The desynchrony of the accumulation processes of total lignin and BnComT1s transcripts in most growth stages indicated that BnCOMT1S could be responsible for the synthesis of a specific subunit of lignIn or that they participate in other pathways such as the melatonin biosynthesis pathway.
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Neural Bases of Unconscious Error Detection in a Chinese Anagram Solution Task: Evidence from ERP Study.

TL;DR: In this article, the authors used event-related potentials (ERPs) in the brain to explore the neural bases of unconscious error detection when subjects solved a Chinese anagram task, and they found that the UED response elicited a more negative ERP component (N2) than did no error (NE) and detect error (DE) responses in the 300-400-ms time window.
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SsNEP2 Contributes to the Virulence of Sclerotinia sclerotiorum

TL;DR: The data suggest that SsNEP2 is involved in fungal virulence by affecting ROS levels in S. sclerotiorum by serving as a pathogen-associated molecular pattern (PAMP) and trigger host pattern triggered immunity to promote the necrotrophic lifestyle of S.