W
Wei Wang
Researcher at Northeast Agricultural University
Publications - 10
Citations - 463
Wei Wang is an academic researcher from Northeast Agricultural University. The author has contributed to research in topics: Transcriptome & Kinase. The author has an hindex of 8, co-authored 10 publications receiving 350 citations.
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H2S induces Th1/Th2 imbalance with triggered NF-κB pathway to exacerbate LPS-induce chicken pneumonia response.
TL;DR: H2S exposure can lead to Th1/Th2 immune imbalance, repress the anti-inflammatory effect of PPAR-γ/HO-1, and then activate NF-κB pathway-related genes and the downstream genes to aggravate pneumonia induced by LPS.
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Ammonia induces Treg/Th1 imbalance with triggered NF-κB pathway leading to chicken respiratory inflammation response.
TL;DR: Results are consistent with transcriptome detection, indicating that ammonia has a negative effect on immune responses and causes inflammatory injury of the trachea, and the molecular mechanism of NH3-PM2.5 induced respiratory diseases is explored.
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Application of transcriptome analysis: Oxidative stress, inflammation and microtubule activity disorder caused by ammonia exposure may be the primary factors of intestinal microvilli deficiency in chicken.
TL;DR: The injurious mechanism of NH3 exposure is perfects and a new insight and method for environmental risk assessment is provided, thus destroying the balance of nutritional transporters.
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Ammonia regulates chicken tracheal cell necroptosis via the LncRNA-107053293/MiR-148a-3p/FAF1 axis
TL;DR: Results indicated that lncRNA-107053293 regulated necroptosis by acting as a competing endogenous RNA of miR-148a-3p.
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H2S inhalation-induced energy metabolism disturbance is involved in LPS mediated hepatocyte apoptosis through mitochondrial pathway.
TL;DR: H2S combined with the LPS aggravated the level of energy metabolism disorders and apoptosis, indicating that H2S inhalation-induced energy metabolism disturbance is involved in LPS-mediated hepatocyte apoptosis through the mitochondrial pathway.