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Wen-Li Qiu
Researcher at Iowa State University
Publications - 4
Citations - 219
Wen-Li Qiu is an academic researcher from Iowa State University. The author has contributed to research in topics: Arabidopsis & Gene silencing. The author has an hindex of 4, co-authored 4 publications receiving 177 citations.
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Journal ArticleDOI
Soybean Homologs of MPK4 Negatively Regulate Defense Responses and Positively Regulate Growth and Development
Jian-Zhong Liu,Heidi D. Horstman,E. J. Braun,Michelle A. Graham,Chunquan Zhang,Duroy A. Navarre,Wen-Li Qiu,Yeunsook Lee,Dan Nettleton,John H. Hill,Steven A. Whitham +10 more
TL;DR: Results indicate that GmMPK4s negatively regulate SA accumulation and defense response but positively regulate plant growth and development, and their functions are conserved across plant species.
Journal ArticleDOI
Positive and negative roles for soybean MPK6 in regulating defense responses.
Jian-Zhong Liu,E. J. Braun,Wen-Li Qiu,Yafei Shi,Francismar Corrêa Marcelino-Guimarães,Duroy A. Navarre,John H. Hill,Steven A. Whitham +7 more
TL;DR: It is shown that silencing of GmMPK6 in soybean using virus-induced gene silencing mediated by Bean pod mottle virus caused stunted growth and spontaneous cell death on the leaves, a typical phenotype of activated defense responses.
Journal ArticleDOI
The MAPK Kinase Kinase GmMEKK1 Regulates Cell Death and Defense Responses
Hui-Yang Xu,Chi Zhang,Zhen-Chao Li,Zhi-Rong Wang,Xu-Xu Jiang,Yafei Shi,Sheng-Nan Tian,E. J. Braun,Yu Mei,Wen-Li Qiu,Sen Li,Bo Wang,Juan Xu,Duroy A. Navarre,Dongtao Ren,Ninghui Cheng,Paul A. Nakata,Michelle A. Graham,Michelle A. Graham,Steven A. Whitham,Jian-Zhong Liu +20 more
TL;DR: The results indicate that GmMEKK1 plays both positive and negative roles in immunity and appears to differentially activate downstream MPKs by promoting Gm MPK6 activation but suppressing GmMPK3 activation in response to flg22.
Journal ArticleDOI
S-Nitrosylation inhibits the kinase activity of tomato phosphoinositide-dependent kinase 1 (PDK1)
TL;DR: Evidence is provided that NO may trigger cell death in tomato by inhibiting the activity of phosphoinositide-dependent kinase 1 (SlPDK1) via S-nitrosylation, establishing a potential link between NO-triggered cell death and inhibition of the kinase activity of tomato PDK1.