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William Cenens

Researcher at University of São Paulo

Publications -  22
Citations -  993

William Cenens is an academic researcher from University of São Paulo. The author has contributed to research in topics: Xanthomonas citri & Gene. The author has an hindex of 14, co-authored 20 publications receiving 770 citations. Previous affiliations of William Cenens include Katholieke Universiteit Leuven.

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Engineered Endolysin-Based “Artilysins” To Combat Multidrug-Resistant Gram-Negative Pathogens

TL;DR: This work modified endolysins by protein engineering to create Artilysins that are able to pass the outer membrane and become active against Pseudomonas aeruginosa and Acinetobacter baumannii, two of the most hazardous drug-resistant Gram-negative pathogens.
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Art-175 is a highly efficient antibacterial against multidrug-resistant strains and persisters of Pseudomonas aeruginosa

TL;DR: Art-175 is a novel antibacterial that is well suited for a broad range of applications in hygiene and veterinary and human medicine, with a unique potential to target persister-driven chronic infections.
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Bacteria-Killing Type IV Secretion Systems.

TL;DR: This review will focus on this special subtype of T4SS by describing its distinguishing features, similar systems in other proteobacterial genomes, and the nature of the effectors secreted by these systems and their cognate inhibitors.
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Expression of a Novel P22 ORFan Gene Reveals the Phage Carrier State in Salmonella Typhimurium

TL;DR: This study is the first to reveal molecular and genetic markers authenticating pseudolysogenic development, thereby exposing a novel mechanism, timing, and populational distribution in the realm of phage–host interactions.
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A Multifaceted Study of Pseudomonas aeruginosa Shutdown by Virulent Podovirus LUZ19

TL;DR: A multifaceted study combining quantitative PCR, microarray, RNA-seq, and two-dimensional gel electrophoresis to obtain a global overview of alterations in DNA, RNA, and protein content in Pseudomonas aeruginosa PAO1 cells upon infection with the strictly lytic phage LUZ19 finds new features of phage infection.